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Mengying Li

As seen in: Color Research & Application, Nature, Wiley Online Library, PLOS Medicine, BMJ Open Diabetes Research & Care, Frontiers in Public Health, Gut Journal, MDPI, Taylor & Francis Online, ACS Publications and
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Articles

DDIT3, OTUB2, and ASS1 regulate arginine biosynthesis in colorectal cancer cells under arginine deficiency

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By Mengying Li, Jie Zhang, Xiaolin Chen, Xiuqun Zou, Jiamin Wang, Wenyan Huang, Haixia Peng, Hao Jia, Zhaoyuan Hou
| Cell Reports Verified
Keywords ASS1 ubiquitination OTUB2 AMPK DDIT3 C/EBPα arginine deprivation therapy ADT Research topic(s) CP: cancer CP: molecular biology Introduction Tumor cells rely on extensive nutrient consumption, such as glucose, nucleic acids, lipids, and amino acids to fuel rapid proliferation, invasion, immune evasion, and angiogenesis.1 Arginine, a semi-essential amino acid, plays dual roles in cellular metabolism: it serves as a critical intermediate in the urea cycle and as a primary substrate for...
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circ-E2F3 drives ovarian cancer progression via the miR-1305/STAT3 axis through modulation of ferroptosis

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By Mengying Li, Limei Lai
| Nature Verified
Abstract Ovarian cancer (OC) remains the most lethal gynecologic malignancy, with approximately 75% of patients experiencing recurrence within 18–24 months and facing poor clinical outcomes. Accumulating evidence demonstrates that circular RNAs (circRNAs) play crucial roles in OC development and that ferroptosis serves as a novel tumor suppressor mechanism. This study aimed to elucidate the functional role and underlying molecular mechanisms of circ-E2F3 in OC progression.
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Global changes in gene expression and splicing in alcoholic liver disease

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By Ilya Blokhin, Derek Van Booven, Josepmaria Argemi, Mengying Li, J. Rao, Estelle Barbier, Markus Heilig, Jin Cheng, Antoine Douaihy, Ramon Bataller, Claes Wahlestedt
| Nature Verified
Abstract Alcohol use disorder is a widespread illness commonly leading to alcoholic liver disease (ALD) and cirrhosis with an increased incidence of hepatocellular carcinoma (HCC), but the mechanisms of alcohol-related oncogenesis in the liver are incompletely understood. We tested the hypothesis that ALD predisposes to HCC via dysregulation of splicing.
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