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Articles by Michael Heide
Molecular genotype-phenotype correlation in ACTB- and ACTG1-related non-muscle actinopathies
Keywords non-muscle actinopathies cytoskeleton cytoskeletal actin ACTB ACTG1 Baraitser-Winter cerebrofrontofacial syndrome ACTB-related dystonia-deafness syndrome hearing loss Introduction The ability to make accurate diagnoses, reliably estimate prognoses, and having a deep understanding of underpinning molecular and disease mechanisms are prerequisites to developing precision medicine approaches for genetic disorders.
Communication as a Key Performance Indicator in Employer Branding in the Context of the Social Economy-A Quantitative Study
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Employer Branding - Employer Attractiveness as a New Mindset in the Healthcare Sector
Alsheyab, M., Filimon, N., & Fusté-Forné, F. (2023). Hospitality management in times of crisis: a corporate social responsibility perspective. International Journal of Islamic and Middle Eastern Finance and Management, 16(5), 873–891. https://doi.org/10.1108/IMEFM-03-2022-0122 Ambler, T., & Barrow, S. (1996). The employer brand. Journal of brand management, 4(3), 185–206. https://doi.org/10.1057/bm.1996.42 Amelung, V. Angelkorte, M., Augurzky, B., Brauer, R.
Genetic Modification of Primate Cerebral Organoids
The procedures described here represent a unified protocol for the generation of cerebral organoids from different primate species with a targeted electroporation approach. This allows the ectopic expression of a GOI in a model system that emulates primate (including human) (patho)physiological neocortex development. This unified protocol for the generation of primate cerebral organoids uses the same materials (e.g., media) and protocol steps for all four primate species presented.
Human-specific ARHGAP11B increases size and folding of primate neocortex in the fetal marmoset
Information Published In Science Volume 369 | Issue 6503 31 July 2020 Copyright Copyright © 2020 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. This is an article distributed under the terms of the Science Journals Default License. Article versions You are viewing the most recent version of this article.
Human-specific ARHGAP11B increases size and folding of primate neocortex in the fetal marmoset
Report View ORCID ProfileMichael Heide1,*, View ORCID ProfileChristiane Haffner1, View ORCID ProfileAyako Murayama2,3, View ORCID ProfileYoko Kurotaki4, View ORCID ProfileHaruka Shinohara4, View ORCID ProfileHideyuki Okano2,3, View ORCID ProfileErika Sasaki4, View ORCID ProfileWieland B. Huttner1,* 1Max Planck Institute of Molecular Cell Biology and Genetics, 01307 Dresden, Germany. 2Department of Physiology, Keio University School of Medicine, Tokyo 160-8582, Japan.
Human-specific ARHGAP11B increases size and folding of primate neocortex in the fetal marmoset
Report View ORCID ProfileMichael Heide1,*, View ORCID ProfileChristiane Haffner1, View ORCID ProfileAyako Murayama2,3, View ORCID ProfileYoko Kurotaki4, View ORCID ProfileHaruka Shinohara4, View ORCID ProfileHideyuki Okano2,3, View ORCID ProfileErika Sasaki4, View ORCID ProfileWieland B. Huttner1,* 1Max Planck Institute of Molecular Cell Biology and Genetics, 01307 Dresden, Germany. 2Department of Physiology, Keio University School of Medicine, Tokyo 160-8582, Japan.
Human-specific ARHGAP11B increases size and folding of primate neocortex in the fetal marmoset
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A tunable refractive index matching medium for live imaging cells, tissues and model organisms
There is a shared sentiment that the work is clever and the advance in terms of improving live 3D imaging is potentially very important, but the results are quite preliminary in terms of thorough quantitative validation, and thus it will require quite a bit of work to bring it up to the level of eLife. We would like to thank the reviewers and the editorial team for a constructive and competent eLife review round.
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