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Articles

Cell surface CD55 traffics to the nucleus leading to cisplatin resistance and stemness by inducing PRC2 and H3K27 trimethylation on chromatin in ovarian cancer | Molecular Cancer | Full Text

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By Babal Kant, J. Mark, Microbiome Analytics, Metabolomics Core
| Molecular Cancer Verified
Cancer cells and others, CP70 (Culture media: DMEM 10% FBS), A2780 (Culture media DMEM: 10% FBS), SKOV3 (Culture media: McCoy5a 10% FBS), TOV112D (Culture media: MCDB 15% FBS), OVCAR8 (Culture media: DMEM 10% FBS), OV81 (Culture media: DMEM 10% FBS), HEK293T (Culture media: DMEM 10% FBS), Jurkat (Culture media: RPMI 10% FBS) cells were grown in recommended growth media in humidified incubator containing 5% CO2.
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Cell surface CD55 traffics to the nucleus leading to cisplatin resistance and stemness by inducing PRC2 and H3K27 trimethylation on chromatin in ovarian cancer | Molecular Cancer

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By J. Mark, Metabolomics Core, Babal Kant, Microbiome Analytics
| link.springer.com
Abstract Platinum resistance is the primary cause of poor survival in ovarian cancer (OC) patients. Targeted therapies and biomarkers of chemoresistance are critical for the treatment of OC patients. Our previous studies identified cell surface CD55, a member of the complement regulatory proteins, drives chemoresistance and maintenance of cancer stem cells (CSCs). CSCs are implicated in tumor recurrence and metastasis in multiple cancers.
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