Neel Shah on Muck Rack

Neel Shah

  • Contributor, Freelance
  • Chief Medical Officer, Maven Clinic
Boston
As seen in: LinkedIn, Medium, MSN, BBC Mundo, HuffPost, Mumbai Mirror, Nature, The Washington Post, TIME, Wiley and
chief medical officer @mavenclinic, founder @marchformoms @costsofcare // building a world where every person can choose to grow their family with dignity
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Articles

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By Neel Shah
| DevOps.com
Agentic SRE is the evolution of site reliability engineering where AI agents help observe systems, reason over telemetry and take bounded operational actions under human-defined guardrails. The goal is not to replace SREs, but to reduce toil, speed up diagnosis and make incident response more consistent and scalable. Modern systems are too distributed, noisy and fast-moving for purely manual operations to keep up.
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By Neel Shah
| DevOps.com
Your SRE team is drowning. Not in downtime or failed deployments — in notifications. According to research from PagerDuty, most incident responders receive over 10 alerts per shift, the vast majority of which require no immediate action. Across a typical enterprise, that volume can exceed 2,000 alerts per week, with only 3% genuinely warranting attention. The rest? Noise. Expensive, demoralizing, burnout-inducing noise. Alert fatigue isn’t a new problem, but in 2026, it’s reaching a breaking point.
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Multiplexed single-cell transcriptomics reveals diverse phenotypic outcomes for pathogenic SHP2 variants

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By Robert A Kozol, Peishi Rao, Yvonne T. Kschonsak, Anne E. van Vlimmeren, Ross M Giglio, Ziyuan Jiang, Minhee Lee, José L McFaline-Figueroa, Neel Shah
| Science Magazine
Abstract INTRODUCTION RESULTS DISCUSSION MATERIALS AND METHODS Acknowledgments Supplementary Materials REFERENCES Information & Authors Metrics & Citations View Options References Figures Tables Media Share Abstract The protein tyrosine phosphatase SHP2, encoded by PTPN11, is an important regulator of Ras/mitogen-activated protein kinase signaling that acts downstream of receptor tyrosine kinases and other transmembrane receptors.
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