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Nipasiri Voraphani on Muck Rack

Nipasiri Voraphani

As seen in: ScienceDirect, The Lancet, Respiratory Medicine
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Articles

Rhinitis, sinusitis, and ocular allergy IL-2 synergizes with proinflammatory type 3 inducers to amplify mixed type 2–type 3 inflammation in nasal polyps

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By Nipasiri Voraphani, Stefano Guerra, Mabel Zawacki
| ScienceDirect Verified
Rhinitis, sinusitis, and ocular allergy IL-2 synergizes with proinflammatory type 3 inducers to amplify mixed type 2–type 3 inflammation in nasal polyps Author links open overlay panelMin Wang PhD a b, Qinqin Zhang MS a, Yifei Wang BS a, Ying Sun MD, PhD b c, Nan Zhang MD, PhD a, Mengdi Liu MS a, Haiyang Meng MS a, Ying Li BS a b, Xiangdong Wang MD, PhD a b d , Luo Zhang MD, PhD a b d , Claus Bachert MD, PhD e Show more Background Chronic rhinosinusitis with nasal polyps involves mixed type...
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Circulating CC16, immune response to Mycoplasma pneumoniae and lung function deficits: a population-based, multi-cohort study

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By Amber Spangenberg, Steven A. Belinsky, Maria A. Picchi, Erik Melén, Nipasiri Voraphani, Inger Kull, Angela Simpson, Clare Murray, Marilyn Halonen, Julie G. Ledford, Stefano Guerra
| Respiratory Medicine Verified
Keywords Asthma CC16 lung function Mycoplasma pneumoniae Get full text access Log in, subscribe or purchase for full access. References 1. Rava, M. ∙ Tares, L. ∙ Lavi, I. ... Serum levels of Clara cell secretory protein, asthma, and lung function in the adult general population J Allergy Clin Immunol. 2013; 132:230-232 2. Shijubo, N. ∙ Itoh, Y. ∙ Yamaguchi, T. ... Serum and BAL Clara cell 10 kDa protein (CC10) levels and CC10-positive bronchiolar cells are decreased in smokers Eur Respir J.
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Circulating CC16 and Asthma: A Population-Based, Multi-Cohort Study from Early Childhood Through Adult Life

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By Use Policies, Nipasiri Voraphani, Debra A. Stern, Julie G. Ledford
Rationale: CC16 is an anti-inflammatory protein highly expressed in the airways. CC16 deficiency has been associated with lung function deficits, but its role in asthma has not been established conclusively. Objectives: To determine 1) the longitudinal association of circulating CC16 with the presence of active asthma from early childhood through adult life and 2) whether CC16 in early childhood predicts the clinical course of childhood asthma into adult life.
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