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Peina Wang

As seen in: Science Magazine, Dove Medical Press
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Articles

Hiding in plain sight

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By Zeina Nehme, Yosuke Sakamachi, Peina Wang
| Science Magazine Verified
Abstract REFERENCES AND NOTES Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Abstract HIV-1–infected CD4+ T cells undergo lineage marker changes to become induced CD8+ T cells that harbor latent HIV-1 (Cai et al., this issue). Related Research Article HIV-1 infection converts CD4+ T cells to HLA class II–restricted CD8+ T cells Science Translational Medicine3 Jun 2026 Access the full article View all access options to continue reading this article.
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Claudin-1 targeting suppresses tumor growth, invasion, and metastasis in patient-derived cholangiocarcinoma models

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By Zeina Nehme, Yosuke Sakamachi, Peina Wang, Marion Müller, Emilie Crouchet, Frank Jühling, Mélissa Messé, Julien Moehlin, Romain Désert, Fabio Del Zompo, Christine Thumann, Joachim Lupberger, Natascha Roehlen, Emanuele Felli, Patrick Pessaux, Aïna Venkatasamy, Patrice Marchand, Roberto Iacone, Markus Meyer Verified, Alberto Toso, Nabeel Bardeesy, Daniela Sia, Jade Brochon, Laura Meiss-Heydmann, Mihaela Alina Onea
| Science Magazine Verified
Editor’s summary Abstract Supplementary Materials REFERENCES AND NOTES Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Editor’s summary Cholangiocarcinoma (CCA) is an aggressive adenocarcinoma that arises from the bile ducts and has limited treatment options. To explore potential therapeutic targets, Nehme et al. investigated the role of claudin-1 (CLDN1), a transmembrane protein expressed in tight junctions, as an oncogenic driver in CCA.
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Toll-like receptor 5 protects against murine lung fibrosis through reduced dysbiosis, and TLR5 deficiency is associated with human IPF

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By Zeina Nehme, Yosuke Sakamachi, Peina Wang, Emma Wiley, Carol S. Trempus, Harrison Jacobs Verified, Alma Solis, Collin Johnson, Xianglin Meng, Salik Hussain, Jay H. Lipinski, David N. O’Dwyer, Thomas Randall, Brian N. Papas, Benjamin G Wu, Yonghua Li, Matthias Kugler, Sanya Mehta, Erica L. Scappini, Seddon Y. Thomas, Jian-Liang Li, Lecong Zhou, Amelia Roselli, Jason Malphurs, Peer W. Karmaus
| Science Magazine Verified
Editor’s summary Abstract Supplementary Materials REFERENCES AND NOTES Information & Authors Metrics & Citations Check Access References Figures Tables Media Share Editor’s summary Susceptibility to idiopathic pulmonary fibrosis (IPF) has been linked to polymorphisms in genes involved in epithelial integrity and host defense, and mouse models have demonstrated the presence of lung dysbiosis preceding development of pulmonary fibrosis.
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