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Richárd Bártfai on Muck Rack

Richárd Bártfai

As seen in: Nature, BioRxiv, Cell Press
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Articles

Unravelling malaria latency: parasite intrinsic and environmental factors influencing dormant liver stages

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By Ruth Zondag, Ellen Ploeger, Clemens H.M. Kocken, Richárd Bártfai
| Cell Press Verified
Keywords hypnozoite Plasmodium transmission relapse signaling Malaria and its dormant stage Malaria is responsible for over 600 000 deaths annually [1]. The disease is caused by apicomplexan parasites of the genus Plasmodium, the six members known to infect humans being Plasmodium falciparum, Plasmodium vivax, Plasmodium ovale curtisi, Plasmodium ovale wallikeri, Plasmodium malariae, and Plasmodium knowlesi [2,3]. P.
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CUT&Tag and DiBioCUT&Tag enable investigation of the AT-rich and dynamic epigenome of Plasmodium falciparum from low input samples.

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By Jonas Gockel, Tobias Spielmann, Richárd Bártfai, Gala Ramon-Zamorano
| BioRxiv
Abstract Phenotypic variation between malaria parasites is one of the major contributors to the pathogens success and is regulated by differences in heterochromatin-mediated gene silencing. Currently, the heterochromatin landscape is mostly profiled utilising chromatin immunoprecipitation followed by next-generation sequencing (ChIP-seq).
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The Plasmodium falciparum histone methyltransferase PfSET10 is dispensable for the regulation of antigenic variation and gene expression in blood stage parasites

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By Matthias T. Wyss, Abhishek Kanyal, Igor Niederwieser, Richárd Bártfai
| BioRxiv
Abstract The malaria parasite Plasmodium falciparum employs antigenic variation of the virulence factor P. falciparum erythrocyte membrane protein 1 (PfEMP1) to escape adaptive immune responses during blood infection. Antigenic variation of PfEMP1 occurs through epigenetic switches in the mutually exclusive expression of individual members of the multi-copy var gene family. var genes are located in perinuclear clusters of transcriptionally inactive heterochromatin.
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