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R.K. Subbarao Malireddi on Muck Rack

R.K. Subbarao Malireddi

As seen in: Cell Press
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Articles

Innate immune and metabolic signals induce mitochondria-dependent membrane lysis via mitoxyperiosis

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By Yaqiu Wang, Jianlin Lu, Alexandre F. Carisey, Sangappa B. Chadchan, R.K. Subbarao Malireddi, Bhesh Raj Sharma, Nagakannan Pandian, Rebecca E. Tweedell, Gustavo Palacios, Nathalie Becerra Mora, Camenzind G. Robinson, Aaron Pitre, Peter Vogel, Taosheng Chen, Michael Murphy, Thirumala-Devi Kanneganti
| Cell Press Verified
Keywords metabolism mitochondria oxidative damage mTOR carbon starvation inflammatory cell death inflammasome innate immunity cytokine tumor Get full text access Log in, subscribe or purchase for full access. References 1. Zhou, R. ∙ Yazdi, A.S. ∙ Menu, P. ... A role for mitochondria in NLRP3 inflammasome activation Nature. 2011; 469:221-225 2. Mills, E.L. ∙ Kelly, B. ∙ Logan, A. ... Succinate Dehydrogenase Supports Metabolic Repurposing of Mitochondria to Drive Inflammatory Macrophages Cell.
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A MyD88/IL1R Axis Regulates PD-1 Expression on Tumor-Associated Macrophages and Sustains Their Immunosuppressive Function in Melanoma

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By Sarang Tartey, Geoffrey Neale, Peter Vogel, R.K. Subbarao Malireddi
| cancerres.aacrjournals.org
Tumor Biology and Immunology Sarang Tartey, Geoffrey Neale, Peter Vogel, R.K. Subbarao Malireddi and Thirumala-Devi Kanneganti DOI: 10.1158/0008-5472.CAN-20-3510 Published May 2021 This article requires a subscription to view the full text. You may purchase access to this article or login to access your subscription using the links below. Abstract Macrophages are critical mediators of tissue homeostasis, cell proliferation, and tumor metastasis.
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A MyD88/IL-1R axis regulates PD-1 expression on tumor-associated macrophages and sustains their immunosuppressive function in melanoma

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By Sarang Tartey, Geoffrey Neale, Peter Vogel, R.K. Subbarao Malireddi
| cancerres.aacrjournals.org
Research Article Sarang Tartey, Geoffrey Neale, Peter Vogel, R.K. Subbarao Malireddi and Thirumala-Devi Kanneganti DOI: 10.1158/0008-5472.CAN-20-3510 Abstract Macrophages are critical mediators of tissue homeostasis, cell proliferation, and tumor metastasis. Tumor-associated macrophages (TAMs) are generally associated with tumor-promoting immunosuppressive functions in solid tumors.
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