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Articles by Rob Wüst
Skeletal muscle properties in long COVID and ME/CFS differ from those induced by bed rest
Abstract Patients with long COVID and myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) suffer from post-exertional malaise. The accompanying physical inactivity may contribute to a lower aerobic capacity and may explain skeletal muscle adaptations in these patients. Here, we compare whole-body exercise responses and skeletal muscle adaptations after strict 60-day bed rest in healthy people with those in long COVID and ME/CFS patients, and healthy age- and sex-matched controls.
Putting the brakes on muscle growth: Myostatin as regulator of disuse atrophy
1 INTRODUCTION Skeletal muscle comprises ∼45% of human body mass and is essential for movement and force production. It is a highly metabolic and plastic tissue that adapts its size and metabolism to environmental and functional demands.
Author Correction: Current status and future perspectives on the mechanistic and pathophysiological understanding of long COVID
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Correction to: Communications Medicine https://doi.org/10.1038/s43856-025-01300-z. published online 29 April 2026 In the version of this article, Micheal Natt (Lived Experience Expert, Coventry, UK; Northwest Lung Centre, Manchester University Hospitals, Manchester, UK) was listed with a third, incorrect affiliation.
By Mark A. Faghy, Rob Wüst, Daniel M. Altmann, Ruth EM Ashton, Sarah McMullen, Rae Duncan, Andrew Ewing, Elke Hausmann, Sanjay Gupta Verified, Mady Hornig, David Joffe, Binita Kane, M. Asad Khan, Micheal Natt, Rebecca Owen, David Putrino, Lindsay Skipper, Claire Taylor, Callum Thomas, David Tuller, Danielle Beckman, Arneaux Kruger, Etheresia Pretorius
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Nature
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Myofiber-specific knockout of TGF-β type I receptors in mice concurrently drives muscle hypertrophy, oxidative metabolism, and absolute force
Abstract Transforming growth factor-β (TGF-β) signaling is associated with progressive skeletal muscle wasting. It is unknown whether myofibre-specific knockout of TGF-β type I receptors affects muscle transcriptome, mass, contractile force and oxidative metabolism.
Current status and future perspectives on the mechanistic and pathophysiological understanding of long COVID
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Abstract Viral and infectious illnesses can exert profound and enduring effects on population health and well-being. In the aftermath of SARS-CoV-2 infection, post-acute sequelae, collectively referred to as Long COVID, have emerged as a major global health challenge, affecting more than 400 million people and contributing to estimated annual economic costs exceeding $1 trillion.
By Mark A. Faghy, Rob Wüst, Daniel M. Altmann, Ruth EM Ashton, Sarah McMullen, Andrew Ewing, Elke Hausmann, Sanjay Gupta, Mady Hornig, David Joffe, Binita Kane, M. Asad Khan, Rebecca Owen, David Putrino, Lindsay Skipper, Claire Taylor, Callum Thomas, David Tuller, Danielle Beckman, Arneaux Kruger, Etheresia Pretorius, Rae Duncan, Micheal Natt
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Nature
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Transfer of IgG from long COVID patients induces symptomology in mice
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Results We enrolled 34 patients from the Amsterdam UMC outpatient post-COVID clinic based on World Health Organization definition. All participants had confirmed prior SARS-CoV-2 infection and were in good physical and mental health prior to infection. None of the individuals were hospitalized for COVID-19, and their symptoms persisted for a minimum of 6 months following the initial infection (Table 1).
By Hung-Jen Chen, Brent Appelman, Hanneke L.D.M. Willemen, Amélie V. Bos, Judith Prado, W. Ashwin Mak, Sabine Versteeg, Mads Larsen, Marije K Bomers, Ayesha H.A. Lavell, Braeden T. Charlton, Rob Wüst, W. Joost Wiersinga, Michèle van Vugt, Gestur Vidarsson, Niels Eijkelkamp, Jeroen den Dunnen, Noa Keijzer, Sara Vieira Goncalves, Chiara.E. Geyer, Eline Schüchner
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Cell Reports Medicine
Verified
Virus-Induced Endothelial Senescence as a Cause and Driving Factor for ME/CFS and Long COVID: Mediated by a Dysfunctional Immune System
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