Robert Cole on Muck Rack

Robert Cole

London
As seen in: LinkedIn, The New York Times, Forbes, Nature, Reuters, The Independent (UK), Wiley Online Library, MDPI, The Globe and Mail, JAMA (The Journal of the American Medical Association) and
Writer, habitat hedger, lime-mortar brickie, family man
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Articles

Enterocytes rely on purine biosynthesis/salvage pathway to facilitate dietary fat absorption

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By Yu Wang, Li Chen, Yingze Ma, Mingqi Zhou, Marcus M. Seldin, Jiangjiang Zhu, Alexander Zak, Xinzhong Dong, Robert Cole, Svetlana Lutsenko, Aleksander Geske
| Nature Verified
Abstract Dietary fat absorption is among the most energy-demanding processes of nutrient uptake. Fatty acid activation, triglyceride synthesis, and the trafficking of chylomicrons through the secretory pathway - all require ATP. How enterocytes accommodate the surge in ATP consumption following fat uptake is unclear. We show that the purine biosynthesis/salvage pathway supplies necessary ATP and that Ankyrin Repeat Domain 9 (ANKRD9) couples ATP synthesis and lipoprotein trafficking.
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Comparative omics profiling reveals differences in biomass, energy production, and vesicle transport between CHO and fast-growing CHL-YN cells - Scientific Reports

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By Yu Tsunoda, Tatiana Boronina, Robert Cole, Noriko Yamano-Adachi, Michael J. Betenbaugh
| Nature Verified
Abstract Chinese hamster lung (CHL)-YN cells are promising novel hosts for producing therapeutic antibodies with the potential to shorten the research, development, and manufacturing timelines in biopharmaceutical production. CHL-YN cells grow twice as fast as Chinese hamster ovary (CHO) cells, with a doubling time of 8.1 h. These cells possess strong glutamine synthetase activity, allowing them to be cultured in glutamine-free media.
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Inhibition of Cardiac p38 Highlights the Role of the Phosphoproteome in Heart Failure Progression

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By Ting Liu, Robert Cole, Sogol Sedighi, Robert OMeally
| biorxiv.org
Abstract Heart failure (HF) is a complex condition characterized by the inability of the heart to pump sufficient oxygen to the organs to meet their metabolic needs. Among the altered signal transduction pathways associated with HF pathogenesis, the p38 mitogen-activated protein kinase (p38 MAPK) pathway, activated in response to stress, has attracted considerable attention for its potential role in HF progression and cardiac hypertrophy.
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