Shibali Das

Editor’s summary Cancer survivors treated with chemotherapy often have long-term side effects, but how chemotherapy affects the heart is not well understood. In mouse models, He et al. found that DNA-damaging chemotherapies such as carboplatin induce cardiac-resident macrophage cell death via apoptosis and necroptosis, which led to repopulation by recruited monocytes.

Highlights • Characterization of the lung landscape of tuberculosis (TB) latency and disease • Lung CD27 + NK cells are a key feature associated with protection during TB latency • TB disease features lung pDCs, IFN-responsive macrophages, and activated T cells Summary Tuberculosis (TB), caused by Mycobacterium tuberculosis ( Mtb) latently infects approximately one-fourth of the world’s population.

Access provided by Correction to: Nature https://doi.org/10.1038/s41586-019-1276-2, published online 05 June 2019. In Fig. 2a of this Letter, owing to an error during the production process, the y-axis label of the right-most graph was incorrectly shown as ‘No. of ILC3s per lung (×103)’ instead of ‘No. of ILC3s per lung (×102)’.