Simonne Longerich on Muck Rack

Simonne Longerich

As seen in: Nature, Cell Press
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Articles

DNA requirement in FANCD2 deubiquitination by USP1-UAF1-RAD51AP1 in the Fanconi anemia DNA damage response

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By Fengshan Liang, Adam Miller, Simonne Longerich, Caroline Tang
| Nature
Abstract Fanconi anemia (FA) is a multigenic disease of bone marrow failure and cancer susceptibility stemming from a failure to remove DNA crosslinks and other chromosomal lesions. Within the FA DNA damage response pathway, DNA-dependent monoubiquitinaton of FANCD2 licenses downstream events, while timely FANCD2 deubiquitination serves to extinguish the response.
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Promotion of RAD51-Mediated Homologous DNA Pairing by the RAD51AP1-UAF1 Complex

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By Adam Miller, Caroline Tang, Fengshan Liang, Simonne Longerich, Olga Buzovetsky
| Cell Press
Association of UAF1 with RAD51 via RAD51AP1   Synergistic Action of RAD51AP1 and UAF1 in RAD51 Recombinase Enhancement   Importance of the RAD51AP1-UAF1 Complex in RAD51 Enhancement   Dependence of DNA Damage Repair and HR on the RAD51AP1-UAF1 Complex We have provided biochemical and genetic data to elucidate the role of the RAD51AP1-UAF1 complex in HR and DNA damage repair (Figure 5Figure 5E).
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