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Articles by Sofia Carlsson
Genetic variation in antidiabetic drug targets: associations with Parkinson’s disease risk and age at onset
Abstract To investigate whether antidiabetic drugs have a biological basis to be repurposed in PD prevention, we applied a drug target Mendelian randomization framework to assess associations between genetic variation in antidiabetic drug targets and PD risk or age at onset (AAO).
Temporal changes and genetic susceptibility to type 2 diabetes (1984-2019; HUNT): a longitudinal, population-based study
Get full text access Log in, subscribe or purchase for full access. References 1. Ong, KL ∙ Stafford, LK ∙ McLaughlin, SA ∙ et al. Global, regional, and national burden of diabetes from 1990 to 2021, with projections of prevalence to 2050: a systematic analysis for the Global Burden of Disease Study 2021 Lancet. 2023; 402:203-234 2. Kaptoge, S ∙ Seshasai, SRK ∙ Sun, L ∙ et al.
Maternal diabetes programs sexually dimorphic early-onset cardiovascular dysfunction in metabolically healthy offspring
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Keywords developmental programming diabetes offspring cardiovascular disease endothelial dysfunction DOHaD inheritance metabolism sex dimorphism Introduction The incidence of cardiovascular disease (CVD) in young individuals has steadily increased in recent decades, in contrast to the decreasing trend in older individuals,1 highlighting the need to further identify risk groups for targeted new preventive measures.
By Allan Z. Zhao, Yuxia Wei, Eftychia Kontidou, Ali Mahdi Verified, Paulo R. Jannig, Sara Torstensson, Hong Jiang, Alice Larsson, Aida Collado Verified, Rawan Humoud, Buket Öztürk Esen, Xiaowei Zheng, Haojiang Lu, Eva Lindgren, Sanjiv Risal, Jian Zhao, Elisabet Stener-Victorin, Henrik Toft Sørensen, Lars Pedersen, John Pernow, Zhichao Zhou, Sofia Carlsson, Sergiu-Bogdan Catrina, Qiaolin Deng, Jacob Grünler, David Ersgård
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Cell Reports Medicine
Verified
Snabb ökning av tidigt debuterande typ 2-diabetes i Sverige
Egenreferat. Förekomsten av typ 2-diabetes ökar globalt, samtidigt som incidensen har planat ut i många höginkomstländer. Den ökande prevalensen drivs i stället till stor del av förbättrad överlevnad hos personer med diabetes. En oroande utveckling är att man i ett flertal länder ser en ökning av tidigt debuterande typ 2-diabetes (diagnos före 40 års ålder, nedan kallad »tidig T2D«) [1].
Type, Etiology, and Duration of Epilepsy as Risk Factors for SUDEP
Glossary GGE= genetic generalized epilepsy; ICD-10= International Classification of Diseases, 10th Revision; IGE= idiopathic generalized epilepsy; NASR= North American SUDEP Registry; OR= odds ratio; SUDEP= sudden unexpected death in epilepsy; TCS= tonic-clonic seizure Footnotes Go to Neurology.org/N for full disclosures. Funding information and disclosures deemed relevant by the authors, if any, are provided at the end of the article. Submitted and externally peer reviewed.
Latent autoimmune diabetes may increase risk for complications vs. type 2 diabetes
Source/Disclosures Disclosures: Carlsson and Wei report no relevant financial disclosures. Please see the study for all other authors’ relevant financial disclosures. You've successfully added Endocrinology: Diabetes to your alerts. You will receive an email when new content is published. Click Here to Manage Email Alerts We were unable to process your request. Please try again later. If you continue to have this issue please contact customerservice@slackinc.com.
Interaction Between GAD65 Antibodies and Dietary Fish Intake or Plasma Phospholipid n-3 Polyunsaturated Fatty Acids on Incident Adult-Onset Diabetes: The EPIC-InterAct Study
Epidemiology/Health Services Research Abstract OBJECTIVE Islet autoimmunity is associated with diabetes incidence. We investigated whether there was an interaction between dietary fish intake or plasma phospholipid n-3 polyunsaturated fatty acid (PUFA) concentration with the 65-kDa isoform of GAD (GAD65) antibody positivity on the risk of developing adult-onset diabetes.
Pharmacologic treatment and SUDEP risk
Abstract Objective We conducted a nationwide case–control study in Sweden to test the hypothesis that antiepileptic drugs (AEDs) mono- or polytherapy, adherence, antidepressants, neuroleptics, β-blockers, and statins are associated with sudden unexpected death in epilepsy (SUDEP) risk. Methods Included were 255 SUDEP cases and 1,148 matched controls. Information on clinical factors and medications came from medical records and the National Patient and Prescription Registers.
Pharmacological treatment and SUDEP risk: A nationwide population-based case-control study
Abstract Objective: We conducted a nationwide case-control study in Sweden to test the hypothesis that antiepileptic drugs (AEDs), mono- polytherapy, adherence, antidepressants, neuroleptics, beta-blockers, and statins are associated with sudden unexpected death in epilepsy (SUDEP) risk. Methods: Included were 255 SUDEP cases and 1148 matched controls. Information on clinical factors and medications came from medical records and the National Patient and Prescription Registers.
Clinical risk factors in SUDEP
Abstract Objective We conducted a nationwide case-control study in Sweden to test the hypothesis that specific clinical characteristics are associated with increased risk of sudden unexpected death in epilepsy (SUDEP). Methods The study included 255 SUDEP cases (definite and probable) and 1,148 matched controls. Clinical information was obtained from medical records and the National Patient Register.
Workers in manufacturing, driving, cleaning jobs at highest type 2 diabetes risk
Swedish adults working in manufacturing, driving and cleaning jobs are three times more likely to develop type 2 diabetes when compared with university teachers and physiotherapists, according to study data presented at the European Association for the Study of Diabetes annual meeting.
Elevations of metabolic risk factors 20 years or more before diagnosis of type 2 diabetes: Experience from the AMORIS study
Table S1. Crude and adjusted Hazard ratios for T2D in relation to metabolic risk factors in the full study population Table S2. 20‐year absolute risk estimates with 95% confidence limits, observed risk and difference between observed and estimated risks. Table S3. Proportions of the study population aged 30–49 years affected by different predicted 20‐year risk levels. Presented overall and by men and women. Table S4.
Elevations of metabolic risk factors 20 years or more before diagnosis of type 2 diabetes - experience from the AMORIS study
Risk factors for type 2 diabetes (T2D) may be present long before diagnosis but few studies have documented the impact of such factors during a pre-diabetic period exceeding 10 years. Here we describe trajectories for metabolic risk factors up to 25 years prior to diagnosis and estimate the absolute 20-year risk for T2D based on a simple set of commonly measured key risk factors.
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