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Songmin Ying on Muck Rack

Songmin Ying

As seen in: Nature
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Articles

PKM2 AP: a micropeptide inhibits vascular calcification via molecular glue-mediated ubiquitination of PKM2

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By Mengqiong Wang, Junjie Cheng, Lin Ma, Jingjuan Yang, Cui Gao, Xin Fang, Ling Ma, Xianghui Fu, Aifu Lin, Songmin Ying, Weiqiang Lin, Yi Yang
| Nature Verified
Abstract Chronic kidney disease (CKD) is a major global public health challenge, and cardiovascular complications such as vascular calcification (VC) are crucial in CKD-related mortality. Emerging evidence shows that open reading frames (ORFs) within non-coding genomic regions can encode micropeptides, but their roles in VC progression remain largely unexplored.
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SREBP2 promotes macrophage alternative activation and allergic airway inflammation independent of cholesterol biosynthesis

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By Yinfang Wu, Kua Zheng, Lingling Dong, Shenwei Gao, Yanping Wu, Zhengyuan Liu, Jieyu Li, Haipin Chen, Qingyu Weng, Miao Li Verified, Chen Zhu, Jiahuan Xu, Songmin Ying, Zhihua Chen
| Nature Verified
Abstract Immunometabolic reprogramming is increasingly recognized as a critical regulator of macrophage activation and function. This study aimed to elucidate the role of the SREBP2-cholesterol biosynthetic pathway in M2 activation of interstitial macrophages (IMs) during allergic asthma. We observed a significant expansion of M2-polarized IMs with heightened SREBP2-cholesterol biosynthetic activity in the lungs of asthmatic mice.
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Lysosomal acidity and cathepsin L activate eosinophils via ARG1-mediated arginine metabolism in allergic airway inflammation - Nature Communications

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By Qingyu Weng, Songmin Ying, Zhihua Chen, Fugui Yan
| Nature Verified
Abstract Eosinophils are the predominant immune cells implicated in the pathogenesis of asthma, highlighting the need for strategies to mitigate their effects. While previous studies have implicated the importance of lysosomes in eosinophil function, the precise role of lysosomes in eosinophil activation during asthma remains insufficiently understood. In this study, we demonstrate that lysosomal acidity and cathepsin L activity in eosinophils are elevated in asthmatic patients and mouse models.
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