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Stephanie Barnes

As seen in: Cell Reports
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Articles

Non-ionotropic signaling through the NMDA receptor GluN2B carboxy-terminal domain drives dendritic spine plasticity and reverses fragile X phenotypes

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By Stephanie Barnes, Aurore Thomazeau, Peter Finnie, Maxwell J. Heinrich, Arnold J. Heynen, Noburu H. Komiyama, Seth Grant, Frank S. Menniti, Emily K. Osterweil, Mark Bear
| Cell Reports Verified
Keywords NMDAR mGluR5 dendritic spines long-term depression LTD fragile X syndrome FXS audiogenic seizures AGS epileptogenesis Glyx-13 non-ionotropic Research topic(s) CP: Neuroscience Introduction N-methyl-D-aspartate receptors (NMDARs) play a crucial role in refining neural circuits that are established during development and modified during experience-dependent learning.
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