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Sukhan Kim

As seen in: Cell Reports, Texas Medical Center
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Articles

Bidirectional control of vesicular GABA release, LTP, and amotivation by GluN2D-selective allosteric modulators and ketamine

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By Viktor Oláh, Isabelle Witteveen, Riley E. Perszyk, Trang Nguyen, Trang Nguyễn, Trạng nguyên, Brianna J Bixler, Detlef Vullhorst, Yeeun Yook, Scott Myers, Elijah Z. Ullman, Hao Xing, Eva Diaz, Sukhan Kim, Nicholas S. Akins, Ken Liu, Chad Camp, Andres Buonanno, Randy Hall, James Q. Zheng, Dennis Liotta, Matthew Kennedy, Katherine W Roche, Shannon L. Gourley, Hongjie Yuan, Stephen F. Traynelis, Alexa M. Leahy, Ryan Keable, Cara T. Khayat, Frances G. Ghinger, John F. Traynelis, Sophia Boraschi, Alet van der Westhuyzen, Matthew J.M. Rowan
| Cell Reports Verified
ArticleVolume 45, Issue 8117707Open access Affiliations & Notes 1Department of Cell Biology, Emory University School of Medicine, Atlanta, GA, 30322, United States of America 2Department of Pharmacology and Chemical Biology, Emory University School of Medicine, Atlanta, GA, 30322, United States of America 3Department of Pediatrics, Emory University School of Medicine, Atlanta, GA, 30322, United States of America 4National Institute of Neurological Disorders and Stroke, Bethesda, MD, 20892,...
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Functional Effects of Disease-Associated Variants Reveal That the S1-M1 Linker of the NMDA Receptor Critically Controls Channel Opening

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By Lingling Xie, Miranda J. McDaniel, Riley E. Perszyk, Sukhan Kim
| Texas Medical Center
Keywords Humans, Epilepsy, Intellectual Disability, Mutation, Missense, Receptors, N-Methyl-D-Aspartate, Glutamate receptors, Channelopathy, GRIN, GluN, Pre-M1, Translational study Abstract The short pre-M1 helix within the S1-M1 linker (also referred to as the pre-M1 linker) between the agonist-binding domain (ABD, S1) and the M1 transmembrane helix of the NMDA receptor (NMDAR) is devoid of missense variants within the healthy population but is a locus for de novo pathogenic variants...
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