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Surawee Chuaiphichai on Muck Rack

Surawee Chuaiphichai

As seen in: Nature, Wiley Online Library
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Articles

iNOS modulates inflammatory responses in an NO-independent manner through direct interaction with IRG1 in mitochondria

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By Marina Diotallevi, Carlos Outeiral, Priyanka A Patel, Gareth Purvis, Surawee Chuaiphichai, Thomas Nicol, Faseeha Ayaz, Daniel A. Nissley, Ganna Krasnoselska, Svenja S. Hester, John McVey, Roman Fischer, Benedikt M. Kessler, Charlotte M. Deane, Raymond Owens, Keith M. Channon, Mark Crabtree
| Nature Verified
Abstract Nitric oxide (NO) has fundamental roles in numerous physiological and pathophysiological processes. In macrophages, NO produced by inducible nitric oxide synthase (iNOS) modulates metabolic changes that are essential to macrophage activation and plasticity, driving the characteristic metabolic switch from oxidative phosphorylation to glycolysis1,2.
Open in Who Shared Issue with article?

Cardiomyocyte tetrahydrobiopterin synthesis regulates fatty acid metabolism and susceptibility to ischaemia-reperfusion injury

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By Sandy Chu, Lisa C Heather, Surawee Chuaiphichai, Thomas Nicol
| Wiley Online Library Verified
Post-ischaemic metabolomic profiles We hypothesised that the protective phenotype observed in cmGch1 KO mice in response to IR injury could be due to altered cardiac metabolism, favouring glycolysis with downregulation of fatty acid oxidation. To investigate the effects of cardiomyocyte-specific deletion of Gch1 on the metabolic response to IR injury, we undertook a non-biased metabolomics analysis, using female mice as the effect on IR injury was more prominent.
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