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Tiepeng Wang

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Author Correction: S -nitrosoglutathione reductase GSNOR drives age-related obesity by promoting adipose tissue whitening through de-nitrosation of Beclin-1

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By Xinhua Qiao, Ting Xie, Yuying Zhang, Chuanxin Sun, Xuanhao Wu, Yuzhe Chen, Qin Yao, Haoyang Shi, Shilong Li, Hongyu Zhao, Tiepeng Wang, Jiao Meng, Li Zhou Verified, Mutian Niu, Yangxi Hu, Hansong Liu, Chang Chen
| Nature Verified
Correction to: Nature Communications https://doi.org/10.1038/s41467-026-69793-3, published online 23 February 2026 In the version of this article initially published, there was a typographical error in Fig. 7b where the label now reading “Beclin-1C351A” appeared originally as “Beclin-1C3561A.” The figure has been amended in the HTML and PDF versions of the article.
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S-nitrosoglutathione reductase GSNOR drives age-related obesity by promoting adipose tissue whitening through de-nitrosation of Beclin-1 - Nature Communications

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By Xinhua Qiao, Ting Xie, Yuying Zhang, Chuanxin Sun, Xuanhao Wu, Yuzhe Chen, Qin Yao, Haoyang Shi, Shilong Li, Hongyu Zhao, Tiepeng Wang, Jiao Meng, Li Zhou Verified, Mutian Niu, Yangxi Hu, Hansong Liu, Chang Chen
| Nature Verified
Abstract Age-related obesity is a growing public health concern linked to various metabolic disorders, yet its underlying mechanisms remain incompletely understood. Here we report that S-nitrosoglutathione reductase (GSNOR), a pivotal denitrosation enzyme, increases in adipose tissue of both male mice and humans from middle-age. GSNOR knockout protects against age-related weight gain and enhances metabolism, whereas adipose-specific GSNOR knock-in mice promotes obesity and metabolic decline.
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