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Vitor Antonio Fortuna on Muck Rack

Vitor Antonio Fortuna

As seen in: Nature, Wiley Online Library
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Articles

PI3K gene variants are associated with elevated IL-6 levels and COVID-19 severe outcomes in a multicenter study

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By Ingrid de Almeida, Gabriella Oliveira, Sara Nunes Vaz, Iris Montaño-Castellón, Daniele Santana, Ricardo Figueiredo, Valdirene Leao Carneiro, Carlos Brites, Vitor Antonio Fortuna, Camila Alexandrina Figueiredo, Soraya Castro Trindade, Helton Estrela Ramos, Bruna Ramos Tosta, Laryssa Cardoso Calmon, Dyjaene de Oliveira Barbosa, Fabiane S. Reis-Goes, Nívia N. Silva
| Nature Verified
Abstract The COVID-19 caused by SARS-CoV-2 has resulted in widely variable clinical outcomes. The virus relies on host metabolic pathways particularly the PI3K/AKT/mTOR axis, which regulate cell survival, metabolism, and viral replication. Genetic variants whitin this pathway may influence disease severity. This study evaluated variants in PI3K genes among 216 mild and 291 severe COVID-19 patients, focusing on rs3730089 (PIK3R1), rs785468 (PIK3R3), and rs11121484 (PIK3CD).
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Subgingival biofilm microbiome in individuals with asthma and periodontitis: Metagenomic analysis

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By Vitor Antonio Fortuna, Isaac S. Gomes-Filho, Giselle R. Pinto, Rogério R. Conceição
| Wiley Online Library Verified
CONFLICT OF INTEREST STATEMENT The authors have no conflicts of interest to declare. Supporting Information Filename Description odi14913-sup-0001-TableS1.docxWord 2007 document , 21.9 KB Table S1. REFERENCES , , , , , , , , , , , , , , , & (2021). Sputum microbiome profiles identify severe asthma phenotypes of relative stability at 12 to 18 months. The Journal of Allergy and Clinical Immunology, 147(1), 123–134. https://doi.org/10.1016/j.jaci.2020.04.018 , , , & (2021).
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Gene expression profile of chronic oral graft-versus-host disease

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By Viviane Almeida Sarmento, Giselle Rocha Pinto, Vitor Antonio Fortuna, Rogério Reis Conceição
| PLOS
Loading metrics Open Access Peer-reviewed Research Article Abstract Among the complications observed after allogeneic hematopoietic stem cell transplantation, graft-versus-host disease (GVHD) is the primary cause of post-transplant mortality. The oral cavity is the second most affected organ target in chronic GVHD. Tissue damage results from the upregulation of inflammatory mediators, which play a critical role in the immunopathogenesis of the disease.
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