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Vladimir Ermakov on Muck Rack

Vladimir Ermakov

As seen in: Wiley Online Library, The Journal of Clinical Investigation

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Articles

Detailed Morphology and Electron Transport in Reduced Graphene Oxide Filled Polymer Composites with a Segregated Structure

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By Vitalii A. Kuznetsov, Vladimir Ermakov, Maksim V. Gudkov, Kseniya A. Shiyanova
| Wiley Online Library Verified
Conflict of Interest The authors declare no conflict of interest. References 1 , , , , Adv. Mater. 2018, 30, 1704386. 2 , , , , , , , , , , , , , , , , Nat. Energy 2018, 3, 148. 3 , , , , Adv. Funct. Mater. 2016, 26, 1678. 4 , , , , , Sens. Actuators, A 2013, 199, 265. 5 , , , , Adv. Sci. 2022, 9, 2201673. 6 , , , , Science 2000, 287, 1801. 7 , , , , ACS Appl. Mater. Interfaces 2021, 13, 43831. 8 , , , , , , J. Mater. Chem. A 2017, 5, 12653. 9 , , , , , , , , , , Composites, Part B 2022, 233, 109639.
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Remote effects of kidney drug transporter OAT1 on gut microbiome composition and urate homeostasis

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By JCI Insight, Vladimir Ermakov, Jeffry C. Granados, Sanjay K. Nigam
| The Journal of Clinical Investigation
Abstract The organic anion transporter OAT1 (SLC22A6, originally identified as NKT) is a multispecific transporter responsible for the elimination by the kidney of small organic anions that derive from the gut microbiome. Many are uremic toxins associated with chronic kidney disease (CKD). OAT1 is among a group of “drug” transporters that act as hubs in a large homeostatic network regulating interorgan and interorganismal communication via small molecules.
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The kidney drug transporter OAT1 regulates gut microbiome-dependent host metabolism

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By JCI Insight, Jeffry C. Granados, Vladimir Ermakov, Koustav Maity
| The Journal of Clinical Investigation
Abstract Organic anion transporter 1 (OAT1/SLC22A6, NKT) is a multispecific drug transporter in the kidney with numerous substrates, including pharmaceuticals, endogenous metabolites, natural products, and uremic toxins. Here, we show that OAT1 regulates levels of gut microbiome–derived metabolites. We depleted the gut microbiome of Oat1-KO and WT mice and performed metabolomics to analyze the effects of genotype (KO versus WT) and microbiome depletion.
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