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Wei Ying on Muck Rack

Wei Ying

As seen in: Line Today (Hong Kong), Nature, arXiv, Oxford Academic, Frontiers, MDPI, Taylor & Francis Online, Science Magazine, The Star (Malaysia), Xinhua News Agency and
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Articles

Effects of Low Battery Warning Sound Types and Driver Extraversion on Range Anxiety in Pure Electric Vehicles

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By Dongyu Yu, Wei Ying, Ruizhen Li
| Taylor & Francis Online
Abstract This study investigates how driver extraversion and low-battery alert sounds (alarm vs. naturalistic) affect range anxiety. Eighty drivers were divided into high and low extraversion groups. During simulated driving, participants received alerts at 10% and 5% battery. Data were collected on eye movements, pupil responses, Primary Appraisal Secondary Appraisal (PASA) cognitive appraisals, energy-saving button reaction times, and System Usability Scale (SUS) ratings.
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Heat-triggered phospholipid flipping stabilizes plasma membrane fluidity

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By Shijun Fan, Peng Gao, Kailai Huang, Wei Ying, Lei Xu, Huan Ye, Yi Xi, Hang Qian, Ting Li, Hua Yuan, Bingtian Ma, Yuping Wang, Jiawei Xiong, Hao Wang, Shiwen Tang, Xuewei Chen, Linfeng Sun, Chengbin Xiang, Shigui Li, Peng Qin, Weilan Chen, Liangzhu Kang
| Nature Verified
Abstract Cells must rapidly counteract heat stress-induced hyperfluidization of the plasma membrane to prevent membrane damage1,2, yet how cells achieve such early protection remains unknown. Here we show that in rice (Oryza sativa), the P4-ATPase OsALA5, together with its β-subunit OsALIS2, mediates a heat-responsive flipping of saturated phosphatidylcholines that rapidly stabilizes plasma membrane fluidity.
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Labile iron starvation in embryonic Kupffer cells aggravates MASH via mitochondrial failure and macrophage dysfunction

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By Ke Wang, Junho Park, Zixuan Zeng, Houji Qin, Andrea Farrell, Whasun Lim, Wei Ying, Garam An, Alex Schlotthauer
| Nature Verified
Abstract Metabolically-dysfunction-associated steatohepatitis (MASH) is characterised by embryonic Kupffer cell (emKC) loss and proinflammatory macrophage infiltration. While iron dysregulation is implicated in MASH, the role of labile iron (Fe2 + ) in mediating emKC survival and function remains unknown. In human and mouse MASLD/MASH livers, emKCs exhibit repressed iron metabolism, reduced labile iron pools, and mitochondrial dysfunction.
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