Xianghua Sun

Abstract Metabolic dysfunction-associated steatotic liver disease (MASLD) is driven by unresolved inflammation, yet precise mechanisms linking immune metabolism to disease progression remain elusive. Here, we identified myeloid-expressed Mas, a G protein-coupled receptor, as a critical metabolic checkpoint in MASLD. Mas expression is elevated in hepatic myeloid cells from patients and diet-induced mouse models.

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CASE REPORT article Volume 19 - 2025 | https://doi.org/10.3389/fnins.2025.1686623 Introduction Central post-stroke pain (CPSP) manifests as persistent or intermittent pain following cerebral infarction or hemorrhage, described as “one of the most agonizing, disabling, and refractory pain syndromes” (Klit et al., 2009; Henry et al., 2008).