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Xiuying Li on Muck Rack

Xiuying Li

As seen in: Color Research & Application, Nature, Wiley Online Library, PLOS, PLOS ONE, Frontiers in Public Health, MDPI, Thorax, Arthritis Research & Therapy, Medscape and
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Articles

Fermentative iron reduction by a psychrotolerant Clostridium -dominant consortium enriched from Antarctic penguin-impacted soils

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By Xin Wang, HONGYAN WANG, Xuhao Wang, Hengyi Liao, Jiaojiao Yang, Huijuan Jin, Min ki Jeon, Yiru Cui, Xiuying Li, Xiaodong Liu, Xin McRae Chen, Li Liao, Yiran Dong, Lisi Jiang, Zongming Xiu, Yi Yang, Erik Hoffnagle
| Nature Verified
Abstract Microbial iron cycling regulates nutrient availability and redox balance in global ecosystems, yet its pathways remain underexplored in ice-free Antarctic terrestrial ecosystems. This study reports the enrichment of a psychrotolerant microbial consortium from penguin-impacted soils on Beaufort Island, Antarctica, capable of reducing Fe(III) to Fe(II) at 4 °C via an anaerobic (likely fermentative) iron-reducing pathway.
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Cofactor binding interactions with reductive dehalogenase shape cobamide utilization in organohalide-respiring bacteria

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By Huijuan Jin, Jingjing Wang, Xiuying Li, Xiaocui Li, Huan He, Ke Ji Shi, Siqi Huang, Frank E. Löffler, Jun Yan, Yiru Cui, Tongyue Zhou
| Nature Verified
Abstract Selective utilization of certain cobamides, the vitamin B12-family enzyme cofactors, is a common physiological trait in organohalide respiration, an essential biogeochemical process relevant to global halogen cycling and the bioremediation of chlorinated contaminants. Variations in cobamide structure, particularly the lower base, can disrupt reductive dehalogenase (RDase) catalysis or even completely inactivate organohalide-respiring bacteria (OHRB).
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TWIST1 drives endothelial-to-mesenchymal-transition to stabilize atherosclerotic plaques - Nature Communications

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By Blanca Tardajos Ayllón, Mannekomba R. Diagbouga, Ankita Das, Siyu Tian, Andreas Edsfeldt, Joanna Kalucka, Jovana Serbanovic-Canic, Emily Chambers, Jiangming Sun, Chrysostomi Gialeli, Mark J. Dunning, Sheila Francis, Xiuying Li, Akiko Mammoto, Michael J. Simons, Helle F. Jørgensen, Isabel Gonçalves, Suowen Xu, Paul Evans
| Nature Verified
Abstract Rupture of unstable atherosclerotic plaques is a major cause of mortality. Endothelial-to-mesenchymal transition associates with advanced atherosclerotic plaques and contributes to plaque progression. We examined the role of Twist1, a transcription factor that drives endothelial-to-mesenchymal transition, in plaque progression by inducible deletion from endothelial cells in hypercholesterolemic mice (Twist1ECKO Apo-/-).
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