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Ying Shan on Muck Rack

Ying Shan

As seen in: Nature, Wiley Online Library, arXiv, PLOS ONE, Frontiers, heart, MDPI, Taylor & Francis Online, Arthritis Research & Therapy, JAMA (The Journal of the American Medical Association) and
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Articles

Glycolytic enolase 1-induced H4K12 lactylation activates quiescent breast cancer stem-like cells in triple-negative breast cancer

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By Shuai Liu, Yunfeng Ma, Yujie Zeng, Dekang Lv, Yaqi Gong, Ruijun Feng, Zihan Wang, Ran Sui, Dan Huang, Yameng Wu, Nuo Chen, Ying Shan, Yuan Huang, Yuesheng Wang, Jinsong Yan, Fei Peng, Quentin Liu, Jie Xu, Zhijie Hou, Junxiu Huang, Mingzhi Yao
| Nature Verified
Abstract Activation of quiescent or extremely slow-cycling breast cancer stem-like cells (qsBCSCs) contributes to tumor progression, yet the regulatory mechanisms involved in triple-negative breast cancer (TNBC) remain elusive. We developed a dual-reporter system combining NANOG-EGFP and H2B-mCherry pulse-chase labeling to separately identify qsBCSCs and active BCSCs (aBCSCs) in vitro and in vivo.
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USP21-mediated deubiquitylation stimulates NuMA recruitment to the cell cortex to promote mitotic spindle orientation

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By Mingzheng Hu, Huixian Ma, Jinqiong Wang, Lin Li, Difei Wang, Hanxiao Yin, Mulin Yang, Feifei Qi, Ying Shan, Huijie Zhao, Jinmin Gao, Dengwen Li, Min Liu, Jun L Zhou, Runa Wang, Zhenqi Yu
| Nature Verified
Abstract Proper orientation of the mitotic spindle is critical for a wide range of biological processes. However, the molecular mechanisms that regulate the formation of the spindle orientation machinery remain incompletely understood. In this study, we identify an essential role for ubiquitin-specific peptidase 21 (USP21)-mediated deubiquitylation in the control of spindle orientation.
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A pilot study of the effect of norepinephrine dose on left ventricular-arterial coupling in patients with septic shock

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By Xueqing Wang, Xuwei Lin, Yingxin Lin, Bin Huang, Qing Feng, Ying Shan, Lei Huang, Yunyin Xu
| Nature Verified
Abstract Left ventricular-arterial (V-A) coupling is a key determinant of cardiovascular performance, which integrates intrinsic myocardial contractility with ventricular afterload. Septic shock frequently disrupts this coupling, and norepinephrine (NE) may worsen it in some scenarios.
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