Ying-Ying Lu

Abstract Autoimmune encephalitis (AE) is an antibody-mediated brain disorder involving self-antibody-driven inflammation. Different subtypes present unique clinical and pathological features, influenced by variations in target antigens and underlying pathophysiological processes. Although the precise pathogenesis of each AE subtype remains incompletely understood, recent research has provided valuable insights into their immunopathological mechanisms.

A set of multipartite orthogonal states is genuinely nonlocal if it is locally indistinguishable in every bipartition of the subsystems. If the set is locally reducible, we say it has genuine nonlocality of type I. Otherwise, we say it has genuine nonlocality of type II. Due to the complexity of the problem, the construction of genuinely nonlocal sets in general multipartite systems has not been completely solved so far.