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Ying-Zhe Cui on Muck Rack

Ying-Zhe Cui

As seen in: The Journal of Clinical Investigation
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Articles

DGAT2 reduction and lipid dysregulation drive psoriasis development in keratinocyte-specific SPRY1-deficient mice

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By JCI Insight, Ying-Ying Li, Li-Ran Ye, Ying-Zhe Cui
| The Journal of Clinical Investigation
ResearchDermatologyInflammationMetabolism Open Access | 10.1172/jci.insight.192507 JCI Insight. https://doi.org/10.1172/jci.insight.192507. Copyright © 2025, Li et al. This work is licensed under the Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. View PDF Abstract Psoriasis is a chronic autoimmune skin disease characterized by abnormal keratinocyte proliferation and immune dysregulation.
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CXCL10 secreted by SPRY1-deficient epidermal keratinocytes fuels joint inflammation in psoriatic arthritis via CD14 signaling

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By Fan Xu, Xing-yu Yang, Yu-Xin Zheng, Ying-Zhe Cui
| The Journal of Clinical Investigation
ResearchDermatologyImmunology Open Access | 10.1172/JCI186135 J Clin Invest. https://doi.org/10.1172/JCI186135. Copyright © 2025, Xu et al. This work is licensed under the Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. View PDF Abstract Psoriatic arthritis (PsA) is a multifaceted chronic inflammatory disease affecting the skin, joints, and entheses, and is a major comorbidity of psoriasis.
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