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Yingying Cui on Muck Rack

Yingying Cui

As seen in: Nature, Wiley Online Library, Frontiers in Immunology, MDPI, Taylor & Francis Online, ACS Publications, Science Magazine, Royal Society of Chemistry, Future Medicine, Academic Radiology
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Articles

A revised Köppen climate classification reveals hydrothermal controls on eco-climate over the Qinghai–Tibet Plateau

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By Tiexi Chen, Zhe Gu, Shengjie Zhou, Xin McRae Chen, Jian Peng, Hongying Li, Wenhui Li, Renjie Guo, Yingying He, Yingying Cui, Deliang Chen, Shengzhen Wang
| Nature Verified
Abstract The Köppen–Geiger climate classification is widely used to represent global eco-climatic patterns, yet its application to high-altitude regions such as the Qinghai–Tibet Plateau (QTP) remains limited because tundra climates (ET) are defined primarily by temperature, overlooking moisture constraints on vegetation.
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A three-dimensional scanning trapped-ion probe

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By Andrea Cimolato, Tahnee Mackensen, Wenqi Pan, Tobias Sägesser, Shreyans Jain, Pavel Hrmo, Alexander Ferk, Matteo Simoni, Yingying Cui, Carmelo Mordini, Daniel Kienzler
| Science Magazine Verified
Abstract INTRODUCTION RESULTS DISCUSSION MATERIALS AND METHODS Acknowledgments Supplementary Materials REFERENCES Information & Authors Metrics & Citations View Options References Figures Tables Media Share Abstract The performance of a wide range of physical systems suffers from undesired fields from nearby surfaces, placing a premium on developing methods for understanding these imperfections.
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Immunological characterization of neuronal intranuclear inclusion disease with kidney injury: an exploratory analysis in a multi-center cohort

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By Ying Ji, Xiaowen Li, Jin Tian, Xian Chen, Guang Ji, Man Xia, Qianru An, Xiang Li, Liangyu Li, Wenjing Song, Ruixue Zhang, Lei Bao, Yuqiao Wang, Yingying Cui, Yuyao Tian, Hao Chen, Maofeng Shi
| Frontiers in Immunology
Abstract Background: Neuronal intranuclear inclusion disease (NIID) is a neurodegenerative disorder caused by GGC repeat expansions in NOTCH2NLC, leading to uN2CpolyG protein deposition. Although immune-mediated renal lesions have been described in NIID, the systemic immunoinflammatory profile associated with kidney injury and its relationship with genetic burden remain undefined.
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