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Yining Qin on Muck Rack

Yining Qin

As seen in: Cell Press
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Articles

Transcription-coupled AID deamination damage depends on ELOF1-associated RNA polymerase II

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By Pengfei Dai, Yuanqing Tan, Yifeng Luo, Tingting Liu Verified, Yanchao Huang, Yafang Shang, Min Huang, Xiaojing Liu, Senxin Zhang, Yanyan Wang, Qian-Xi Li, Niu Li, Lulu Li, Yining Qin, Junqi Liu, Liu Liu, Xia Xie, Yanni Cai, fei chen, Xiaoqi Zheng, Leng-Siew Yeap, Jian Wang, Jinchuan Hu, Fei-Long Meng
| Cell Press Verified
Keywords class switch recombination somatic hypermutation transcription-coupled repair transcription-coupled damage AID ELOF1 Get full text access Log in, subscribe or purchase for full access. References 1. Alt, F.W. ∙ Zhang, Y. ∙ Meng, F.L. ... Mechanisms of programmed DNA lesions and genomic instability in the immune system Cell. 2013; 152:417-429 2. Honjo, T. ∙ Kinoshita, K. ∙ Muramatsu, M. Molecular mechanism of class switch recombination: linkage with somatic hypermutation Annu. Rev. Immunol.
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Taming AID mutator activity in somatic hypermutation

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By Fei-Long Meng, Yining Qin
| Cell Press Verified
Highlights Activation-induced cytidine deaminase (AID)-single-strand (ss)DNA binding mode determines both microscale WRC preference and mesoscale complementarity determining region (CDR) preference; apolipoprotein B editing complex (APOBEC3)A uses a distinct binding mode and shows different DNA sequence preferences. The flexible ssDNA are AID-favored substrates in vitro and in vivo.
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