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Yinshui Chang on Muck Rack

Yinshui Chang

As seen in: Nature, Wiley, Science Magazine, The Journal of Immunology
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Articles

TGF-β and IL-2 differentially shape T follicular regulatory cell differentiation and stability in vitro

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By Luisa Bach, Yinshui Chang, Lisa Maria Steinheuer, Teresa Tromm Steffen, Elena De Domenico, Thomas Ulas, F. Thomas Wunderlich, Marc Beyer, Kevin Thurley, Dirk Baumjohann, Olin Arteaga Transito, Mohammadamin Ghasemi
| Nature Verified
Abstract T follicular helper (Tfh) cells and T follicular regulatory (Tfr) cells play critical roles in regulating the activity of the germinal center (GC), which is essential for the generation of high-affinity antibodies. In the GC, Tfh cells help B cells to proliferate and to differentiate into memory B cells and long-lived plasma cells.
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Genomic deletion of Bcl6 differentially affects conventional dendritic cell subsets and compromises Tfh/Tfr/Th17 cell responses - Nature Communications

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By Hongkui Xiao, Isabel Ulmert, Ilia Kurochkin, Yinshui Chang, Urs Mörbe, Carlos-Filipe Pereira, Barbara U. Schraml, Dirk Baumjohann, Signe Holst
| Nature Verified
Abstract Conventional dendritic cells (cDC) play key roles in immune induction, but what drives their heterogeneity and functional specialization is still ill-defined. Here we show that cDC-specific deletion of the transcriptional repressor Bcl6 in mice alters the phenotype and transcriptome of cDC1 and cDC2, while their lineage identity is preserved.
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An e-Xist-ential two-edged sword for women

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By Yinshui Chang, Byron Hayes, Marco De Giovanni, Zhenrui Zhang
| Science Magazine Verified
Editors' Choice FOCIS HIGHLIGHTS Zhenrui Zhang and Shiv Pillai https://orcid.org/0000-0002-9905-875X pillai@helix.mgh.harvard.eduAuthors Info & Affiliations Abstract Xist-containing ribonucleoproteins drive autoimmunity in women. SIGN UP FOR THE SCIENCEADVISER NEWSLETTER The latest news, commentary, and research, free to your inbox daily For decades, many attempts have been made to better understand the very strong female bias toward the development of autoimmunity.
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