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Yuuki Ohara

As seen in: Wiley Online Library, BioRxiv, Cell Press
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Articles

ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic reprogramming in pancreatic cancer

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By Yuuki Ohara, Huaitian Liu, Amanda Craig, Shouhui Yang
| Wiley Online Library Verified
1 INTRODUCTION Pancreatic cancer is a highly lethal cancer with a 5-year survival of only 12%.1 Pancreatic ductal adenocarcinoma (PDAC) is the most common type of pancreatic cancer, accounting for more than 95% of all malignancies in the pancreas.2 Earlier studies identified several molecular subtypes of PDAC with differences in biological characteristics and patient survival.3-5 Bailey et al.
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LMO3 is a suppressor of the basal-like/squamous PDAC subtype and reduces disease aggressiveness of pancreatic cancer through glycerol 3-phosphate metabolism

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By Yuuki Ohara, Amanda Craig, Huaitian Liu, Shouhui Yang
| BioRxiv
Abstract Pancreatic ductal adenocarcinoma (PDAC) encompasses diverse molecular subtypes, including the classical/progenitor and basal-like/squamous subtypes, each exhibiting distinct characteristics, with the latter known for its aggressiveness. We employed an integrative approach combining transcriptomic and metabolomic analyses to pinpoint potential genes contributing to the basal-like/squamous subtype differentiation.
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ELAPOR1 induces the classical/progenitor subtype and contributes to reduced disease aggressiveness through metabolic reprogramming in pancreatic cancer

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By Yuuki Ohara, Amanda Craig, Huaitian Liu, Shouhui Yang
| BioRxiv
Abstract Pancreatic ductal adenocarcinoma (PDAC) is a heterogeneous disease with distinct molecular subtypes classified as classical/progenitor and basal-like/squamous. We hypothesized that integrative transcriptomic and metabolomic approaches can identify candidate genes whose inactivation contributes to the development of the aggressive basal-like/squamous subtype.
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