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Zhi Jian Wang on Muck Rack

Zhi Jian Wang

As seen in: British Journal of Cancer, Nature, Wiley Online Library, PLOS, PLOS ONE, BMJ Open, Frontiers, ACS Publications, Brookings Institution, Science Magazine and
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Articles

Machine Learning-Assisted Label-Free SERS Decoding of Mitochondrial Molecular Dynamics in Ovarian Granulosa Cells during Aging Click to copy article link Article link copied!

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By Hongjin Xue, Zhanfeng Li, Zhi Jian Wang
| ACS Publications
Cite Jump to ExpandCollapse ArticleJuly 6, 2026 Machine Learning-Assisted Label-Free SERS Decoding of Mitochondrial Molecular Dynamics in Ovarian Granulosa Cells during Aging Click to copy article linkArticle link copied! Hongjin Xue Hongjin Xue Guangdong Key Laboratory of Biomedical Measurements and Ultrasound Imaging, School of Biomedical Engineering, Shenzhen University Medical School, Shenzhen University, Shenzhen 518060, China Medical Engineering and Technology Research Center, School of...
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Anion-Directed Assembly of Atomically Precise Silver Nanofibers: Tunable Inner Diameters and Mechanical Exfoliation into Subnanometer Nanofibers Click to copy article link Article link copied!

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By Rakesh Gupta, Yan Chen, Zhi Jian Wang
| ACS Publications
Abstract Click to copy section linkSection link copied! Nanofibrous architectures, a prominent class of one-dimensional nanomaterials, continue to captivate researchers in the exploration of novel nanofibrous materials and their diverse applications in nanotechnology. Creating crystalline nanofibers with inorganic walls capped by organic ligands is both critical and challenging, requiring precise structural control and consistent internal diameters.
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Mouse model of X-linked Alport syndrome with K229X mutation in the COL4A5 gene

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By Ran Zhang, Weina Jiang, Xuyan Liu, Zhi Jian Wang, Leping Shao, Pei Jin Bai, Shuimi Zhang
| Nature Verified
Abstract X-linked Alport syndrome (XLAS) is a hereditary glomerular basement membrane (GBM) disease caused by COL4A5 mutations, leading to end-stage renal disease. With unclear pathogenesis and limited treatments, reliable animal models are urgently needed. In this study, the mutation K229X in COL4A5 detected in XLAS patients was introduced into mice model by CRISPR/Cas.
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