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Adam Boulton on Muck Rack

Adam Boulton

London
As seen in: Times Radio, MSN UK, Daily Mail, MSN South Africa, The Telegraph, Sky News, The London Standard, BMJ Open, Emergency Medicine Journal, Substack, The Sunday Times and
I RT what amuses or interests me inc. criticism. RT is not an endorsement. I mute or block trolls.
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Articles

Transient Receptor Potential Vanilloid 4 in Macrophages Mediates TGF-β Activation to Drive Myofibroblast Differentiation and Pulmonary Fibrosis

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By Lisa Grove, Caitlin Snyder, Adam Boulton
| The Journal of Biological Chemistry Verified
The authors have declared that no conflict of interest exists. One Sentence Summary: TRPV4 activates TGF-β in macrophages to drive fibrosis. INTRODUCTION Fibrotic interstitial lung diseases (fILDs) are devastating progressive disorders that carry a high mortality and lack a cure (1, 2). The hallmark of fILDs is accumulation of pathogenic fibroblasts referred to as myofibroblasts (3).
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The mechanosensitive channel TRPV4 inhibits pulmonary inflammation by limiting NF-κB signaling in alveolar macrophages

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By Qianqian Hu, Adam Boulton, Nicholas Venetos, Megan E. Grund, Yuxin Wang, Erica Orsini, Yan Liu, Susamma Abraham, Lisa Grove, Ryan J Musich, Caitlin Snyder, Mitchell A. Olman, Rachel G. Scheraga, Haley Ricci, Amber Cardani-Boulton, Vidula Vachharajani
| Science Magazine Verified
Editor’s summary Bacterial lung infection stimulates NF-κB signaling in alveolar macrophages, causing them to produce proinflammatory cytokines that damage the tissue. Boulton et al. found that the mechanosensitive channel TRPV4 in alveolar macrophages suppressed lung inflammation during bacterial pneumonia in mice.
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Retraction

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By Qianqian Hu, Adam Boulton, Nicholas Venetos, John Foley Verified
| Science Magazine Verified
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