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Avais M. Daulat on Muck Rack

Avais M. Daulat

As seen in: Nature, Wiley Online Library, Cell Press
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Articles

CRL2 FEM1B uses heme to recruit BACH1 for degradation and regulate ferroptosis in lung cancer

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By Bashir Ahmed, Daniele Salaun, Jack Jordan, Avais M. Daulat, Chiara Pastorello, Tarcísio Brito, Yuying Yang, Emmanuelle Josselin, Deborah A. Byrne, Stéphane Betzi, Amandine Pelletier, Julien Vernerey, Lorene Ferreira, Rémy Castellano, Stéphane Audebert, Luc Camoin, Attilio Pane, Cherine BECHARA, Jean-Paul Borg, Mauro Modesti, Luca Lignitto
| Cell Press Verified
Keywords ubiquitin proteasome BACH1 FEM1B CUL2 Cullin-RING ubiquitin ligase lung cancer NSCLC heme ferroptosis Introduction Non-small cell lung cancers (NSCLCs) frequently increase iron demand to sustain their growth, which makes them more susceptible to ferroptosis, an iron-dependent cell-death mechanism driven by oxidative stress and peroxidation of polyunsaturated fatty acids (PUFAs).1,2,3,4 Given its role, ferroptosis is increasingly recognized as a critical tumor-suppressor mechanism and...
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The Tumor Suppressor SCRIB is a Negative Modulator of the Wnt/β‐Catenin Signaling Pathway

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By Avais M. Daulat, Monica Wagner, Alexandra Walton, Emilie Baudelet
| Wiley Online Library Verified
Research Article The Tumor Suppressor SCRIB is a Negative Modulator of the Wnt/β‐Catenin Signaling Pathway Avais M.
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ECT2 associated to PRICKLE1 are poor-prognosis markers in triple-negative breast cancer

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By Avais M. Daulat, Pascal Finetti, Monica Wagner, Diego Revinski
| Nature Verified
Abstract Background Triple-negative breast cancers (TNBC) are poor-prognosis tumours candidate to chemotherapy as only systemic treatment. We previously found that PRICKLE1, a prometastatic protein involved in planar cell polarity, is upregulated in TNBC. We investigated the protein complex associated with PRICKLE1 in TNBC to identify proteins possibly involved in metastatic dissemination, which might provide new prognostic and/or therapeutic targets.
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