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Barbara Kronsteiner on Muck Rack

Barbara Kronsteiner

As seen in: Nature, Wiley Online Library, Science Magazine, medRxiv, The Journal of Clinical Investigation, The Journal of Immunology
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Articles

Mucosal-associated invariant T (MAIT) cells are reduced and dysfunctional in acute melioidosis

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By Fazle Chowdhury, Priyanka A. Abraham, Jennifer Hill, Rokibul Hasan, Ayako Kurioka, Patpong Rongkard, Direk Limmathurotsakul, Nicholas Day, Parinya Chamnan, Paul Klenerman, Chris Willberg, Barbara Kronsteiner, Susanna J. Dunachie, Martha Zewdie, Srija Moulik, Hadiza Adebowale, Lovely Barai
| Nature Verified
Abstract Burkholderia pseudomallei (BP), the causative agent of melioidosis, is a major cause of sepsis in Southeast Asia, especially in people with diabetes mellitus (DM). The role of Mucosal-associated invariant T (MAIT) cells; innate-like T cells important for antibacterial immunity; in melioidosis is unknown.
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Iron deficiency causes aspartate-sensitive dysfunction in CD8+ T cells - Nature Communications

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By Megan Teh, Nancy Gudgeon, Joe Frost, Linda Sinclair, Alastair Smith, Christopher L. Millington, Barbara Kronsteiner, Bryan P. Marzullo, Alexandra Preston, Thomas A. Milne, Daniel A. Tennant, Susanna J. Dunachie, Andrew Armitage, Sarah Dimeloe, Victoria Stavrou
| Nature Verified
Abstract Iron is an irreplaceable co-factor for metabolism. Iron deficiency affects >1 billion people and decreased iron availability impairs immunity. Nevertheless, how iron deprivation impacts immune cell function remains poorly characterised. We interrogate how physiologically low iron availability affects CD8+ T cell metabolism and function, using multi-omic and metabolic labelling approaches.
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Dysregulated immunologic landscape of early host response in melioidosis

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By JCI Insight, Patpong Rongkard, Lu Xia, Barbara Kronsteiner
| The Journal of Clinical Investigation
ResearchImmunologyInfectious disease Open Access | 10.1172/jci.insight.179106 JCI Insight. https://doi.org/10.1172/jci.insight.179106. Copyright © 2024, Rongkard et al. This work is licensed under the Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.
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