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Baris Alten

As seen in: Nature, BioRxiv, Cell Press, Cell Reports
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Articles

Lithium as a therapeutic strategy for SNAP25 encephalopathies via modulation of homeostatic plasticity

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By Burak Uzay, Z. Zack Ma, Baris Alten, Lisa M. Monteggia, Ege T. Kavalali, Ihsan Alp Uzay
| Nature Verified
Abstract Synaptic vesicle fusion is orchestrated by the soluble N-ethylmaleimide–sensitive factor attachment receptor (SNARE) complex. The SNARE complex includes SNAP25, synaptobrevin-2, and syntaxin-1. This machinery drives action potential-evoked release, as well as the majority of spontaneous release.
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Cryo-electron tomography reveals postsynaptic nanoblocks in excitatory synapses for transcellular alignment

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By Rong Sun, Mariam Haider, Baris Alten, Liana Wilson
| BioRxiv
Thank you for your interest in spreading the word about bioRxiv. NOTE: Your email address is requested solely to identify you as the sender of this article. Your Email * Your Name * Send To * Enter multiple addresses on separate lines or separate them with commas. Message Subject (Your Name) has forwarded a page to you from bioRxiv Message Body (Your Name) thought you would like to see this page from the bioRxiv website.
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Presynaptic mechanisms underlying GABAB-receptor-mediated inhibition of spontaneous neurotransmitter release

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By Baris Alten, Natalie J. Guzikowski, Zack Zurawski, Heidi E. Hamm
| Cell Reports Verified
Highlights • Scavenging Gβγ prevents GABAB-mediated inhibition of spontaneous release • Regulation of spontaneous glutamate release involves Gβγ-SNARE interactions • Synaptotagmin-1 modulates Gβγ-SNARE interactions for spontaneous glutamate release • Regulation of spontaneous GABA release utilizes Gβγ through alternative targets Summary Inhibition of neurotransmitter release by neurotransmitter substances constitutes a fundamental means of neuromodulation.
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