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Benjamin D. Singer on Muck Rack

Benjamin D. Singer

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Articles

A Daily Clinical Parameters and Outcomes Adjudication Dataset for Critically Ill Patients with Suspected Pneumonia

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By Catherine Gao, Nikolay S. Markov, Mengjia Kang, Luke V. Rasmussen, Wan-Ting Liao, Anna-Maya Pawlowski Verified, Vijeeth Guggilla, Helen Donnelly, Chiagozie A. Pickens, Nandita R. Nadig, Thomas Stoeger, Dan Schneider, Justin Starren, Theresa L. Walunas, G. R. Scott Budinger, Richard G. Wunderink, Alexander V. Misharin, Benjamin D. Singer, Prasanth Nannapaneni, Rebecca K. Clepp
| Nature Verified
Abstract Many analyses of critically ill patients focus on admission features and discharge outcomes, overlooking daily events and intercurrent complications. Pneumonia is a syndromic entity that is difficult to adjudicate outcomes without clinical expertise. Here, we examine clinical features on a day-by-day basis, mirroring the practice of daily multidisciplinary rounds, with annotated pneumonia episodes and outcomes.
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Mitochondrial l -2-hydroxyglutarate is a physiological signalling metabolite

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By Ram P. Chakrabarty, Yuki Aoi, Taylor A Poor, Gregory S. McElroy, Karthik Vasan, Shimaa Soliman, Marta Iwanaszko, Rogan A. Grant, Benjamin Howard, Colleen R. Reczek, Anjali Chandel, Michael D. Kahl, Zhaofa Xu, Kathryn A. Helmin, Qiushi Jin, Dongmei Wang, Peng Gao, Jenna Blum, Zachary Sebo, Feng Yue, Shawn M. Davidson, Steven Gygi, Samuel E. Weinberg, Benjamin D. Singer, Seung Hye Han, Ali Shilatifard, Navdeep S. Chandel, Jonathan G. Van Vranken, Yongchao C. Ma
| Nature Verified
Abstract l-2-Hydroxyglutarate (l-2-HG) is a low-abundance metabolite in mammals because the mitochondrial enzyme l-2-HG dehydrogenase (L2HGDH) oxidizes l-2-HG to 2-oxoglutarate (2-OG) to prevent its accumulation1. In humans, a lack of L2HGDH activity leads to l-2-HG accumulation and causes l-2-hydroxyglutaric aciduria2. Thus, l-2-HG is often classified as a toxic metabolite2,3,4,5. However, whether l-2-HG has any physiological function is unclear.
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Umbilical cord blood cell transcriptional and methylation signatures at birth are associated with BPD development and chorioamnionitis: a pilot study

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By Qianli Liu, Kathryn A. Helmin, Jeanette Bailey, Xóchitl G. Pérez-Leonor, Duc Phan, Hiam Abdala-Valencia, Leena B. Mithal, Benjamin D. Singer, Mary Robbins, Marta Perez Verified, Marta Gil Pérez
| Nature Verified
Abstract Bronchopulmonary dysplasia (BPD) is the most common respiratory disease in preterm infants born at less than 28 weeks gestation. Most existing clinical prediction models for BPD show limited accuracy in predicting BPD development when validated using external data, stressing the need for novel biomarkers to identify at-risk infants for early and effective interventions.
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