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Author Correction: Albumin orchestrates a natural host defence mechanism against mucormycosis - Nature

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By Ioannis Morianos, Vassilis Nidris, Ángeles López-López, Irene Kyrmizi, Sandra M. Camunas-Alberca, Toine Mercier, Yuri Vanbiervliet, Dimitrios Samonakis, Carlos Lax, Agostinho Carvalho, Shivaprakash Mandya Rudramurthy, Kyriakos Petratos, Victoriano Garre, Coral Barbas, Ashraf Ibrahim
| Nature Verified
Correction to: Nature https://doi.org/10.1038/s41586-025-09882-3 Published online 7 January 2026 In the version of this article initially published, Sandra M. Camunas-Alberca was listed without a middle initial; the name is now amended in the HTML and PDF versions of the article.
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Albumin orchestrates a natural host defence mechanism against mucormycosis - Nature Original

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By Ioannis Morianos, Vassilis Nidris, Ángeles López-López, Irene Kyrmizi, Sandra M. Camunas-Alberca, Toine Mercier, Yuri Vanbiervliet, Dimitrios Samonakis, Carlos Lax, Agostinho Carvalho, Shivaprakash Mandya Rudramurthy, Kyriakos Petratos, Victoriano Garre, Coral Barbas, Ashraf Ibrahim
| Nature Verified
Abstract Mucormycosis is an emerging, life-threatening human infection caused by Mucorales fungi1,2,3. Metabolic disorders uniquely predispose an ever-expanding group of patients to mucormycosis through poorly understood mechanisms1,2,4,5, suggesting that uncharacterized host metabolic effectors may confer protective immunity against this infection. Here we uncover a master regulatory role of albumin in host defence against Mucorales through the modulation of fungal pathogenicity.
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Imidazole propionate is a driver and therapeutic target in atherosclerosis - Nature

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By Annalaura Mastrangelo, Iñaki Robles-Vera, Diego Mañanes, Miguel Ladero Galán, Ana Devesa, Alessia Ferrarini, Alberto Benguría, Ana Dopazo, Coral Barbas, Jesús Vázquez Verified, Juan Lopez, Juan López, Konstantinos Stellos, Göran Bergström, Borja Ibáñez, Sophie Ivarsson
| Nature Verified
Abstract Atherosclerosis is the main underlying cause of cardiovascular diseases. Its prevention is based on the detection and treatment of traditional cardiovascular risk factors1. However, individuals at risk for early vascular disease often remain unidentified2. Recent research has identified new molecules in the pathophysiology of atherosclerosis3, highlighting the need for alternative disease biomarkers and therapeutic targets to improve early diagnosis and therapy efficacy.
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