Eng Eong Ooi on Muck Rack

Eng Eong Ooi

Asia–Pacific (APAC)
As seen in: Nature, PLOS Biology, Science Magazine, EBioMedicine, The Lancet, eLife
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Articles

Aminoglycosides induce immune activation but is insufficient for systemic viral control: a double-blind randomized-controlled clinical trial

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By Ayesa Syenina, Yvonne Chan, Dorothy Ng, Esther S. Gan, Jia Yee, Christine YL Tham, Eugenia Z. Ong, Eng Eong Ooi, Shirin Kalimuddin, Punitha Sivanandam
| Nature Verified
Abstract Mucosally-applied aminoglycosides have demonstrated host-directed antiviral activity in preclinical models through induction of interferon-stimulated genes (ISGs). However, it remains unknown if systemically administered aminoglycosides elicit comparable effects in humans.
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Dengue infection elicits skin tissue-resident and circulating CD8 + T cells associated with protection from hospitalization

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By Zhen He, Yudai Sato, Sophie R Shoemaker, Ka-Wai Cheung, Valerie Chew, Michaela Gregorova, Eugenia Ziying Ong, Kuan Rong Chan, Tun-Linn Thein, Yee-Sin Leo, Eng Eong Ooi, Laura Rivino, Noor Zayanah Hamis, Justin SG Ooi
| Science Magazine Verified
Abstract Dengue is spreading globally, and there is urgent need to define immune correlates of protection for this disease. Dengue infection first occurs in the skin following the bite of an infected mosquito; however, knowledge of host immune responses within this site remains sparse. We investigated the phenotypic, functional, and transcriptional profiles of skin and blood T cells in 73 patients with dengue and 10 healthy volunteers.
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Reduced dengue virus pre-membrane protein-HMGB1 interaction activates host cell transcription to attenuate infection - npj Vaccines

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By Tanamas Siriphanitchakorn, Summer Zhang, Dorothy Ng, Xin Yi Yap, Jia Yee, Qiao Zheng, Yi Tan, Eugenia Z. Ong, Kuan Rong Chan, Milly M. Choy, Eng Eong Ooi, Justin S. G. Ooi
| Nature Verified
Abstract To successfully complete an infection lifecycle, viruses have to avoid activating host antiviral and transcriptional responses to avoid disrupting cellular processes and cause premature host cell death. How the host transcriptional response to dengue virus (DENV) infection and its impact on pathogenesis, however, remain unclear.
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