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A new study reveals histidine as a readily measurable and potentially modifiable metabolic biomarker for cancer risk stratification and treatment optimization, but it also highlights the challenges of translating such associations into clinically usable interventions.
ArticleOnline nowOpen access Affiliations & Notes 1Laboratory of Tumor Microenvironment and Therapeutic Resistance, Department of Oncology, KU Leuven, Leuven, 3000, Belgium 2Cell Death Research and Therapy Group, Department of Cellular and Molecular Medicine, KU Leuven, Leuven, 3000, Belgium 3VIB Center for Cancer Biology Research, Leuven, 3000, Belgium 4Department of Applied Computational Cancer Research, Institute for AI in Medicine (IKIM), University Hospital Essen, Essen, 45147, Germany...
Abstract Recent advances in mitochondrial network dynamic and signalling highlight mitochondria as key therapeutic targets across diverse diseases. Yet, high drug development failure rates reflect an incomplete understanding of upstream molecular regulators of mitochondrial fate. Here, we address this gap by reverse engineering of the BH3-only protein BNIP3.
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