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Glenda M Halliday on Muck Rack

Glenda M Halliday

As seen in: Color Research & Application, Nature, Wiley, Wiley Online Library, The BMJ, ACS Publications, Medscape, Science Magazine, Cell Press, The Lancet and
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Articles

Astrocytic lipid dysregulation as an early driver of neurodegeneration

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By Woojin Kim, Glenda M Halliday
| Nature Verified
Abstract Astrocytes have traditionally been cast as supportive glia, but they are increasingly recognized as metabolic hubs that regulate cholesterol synthesis, fatty acid detoxification, lipid droplet dynamics and redox homeostasis in the CNS. Neurons have a limited intrinsic capacity for lipid storage and detoxification and rely heavily on astrocytes to maintain a safe lipid environment.
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Alpha-Synuclein in Neurodegeneration: From Shared Biology to Disease-Specific Phenotypes

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By Feifei Su, Woojin Kim, Glenda M Halliday
| preprints.org
Submitted: 10 February 2026 You are already at the latest version Alpha-synuclein (αSyn) is one of the most abundant proteins in the nervous system and is currently associated with devastating synucleinopathies, yet its biology extends far beyond this. In this review, we outline a unified model suggesting that αSyn‑driven disease emerges within specific neural circuits through the combined effects of cell‑type‑specific roles, subcellular environments, and post‑translational modifications.
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Biological effects of pathologies in Lewy body diseases: why timing matters

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By Elie Matar, Glenda M Halliday
| The Lancet Verified
Get full text access Log in, subscribe or purchase for full access. References 1. Höglinger, GU ∙ Adler, CH ∙ Berg, D ∙ et al. A biological classification of Parkinson's disease: the SynNeurGe research diagnostic criteria Lancet Neurol. 2024; 23:191-204 2. Simuni, T ∙ Chahine, LM ∙ Poston, K ∙ et al. A biological definition of neuronal α-synuclein disease: towards an integrated staging system for research Lancet Neurol. 2024; 23:178-190 3. Mestre, TA ∙ Fereshtehnejad, SM ∙ Berg, D ∙ et al.
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