Gregory Kenneth Muliawan on Muck Rack

Gregory Kenneth Muliawan

As seen in: The Journal of Clinical Investigation
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Articles

Adipocyte-derived FABP4 promotes metabolism-associated steatotic liver–induced hepatocellular carcinoma by driving ITGB1-mediated β-catenin activation

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By Shilpa Gurung, Gregory Kenneth Muliawan, Shakeel Khan, Xue Wu
| The Journal of Clinical Investigation
Abstract Metabolic dysfunction–associated steatotic liver disease–induced (MASLD-induced) hepatocellular carcinoma (HCC) is an emerging malignancy linked to excessive accumulation of adipose tissue and hepatic fat. Understanding the role of adipocytes in the development of MASLD-induced HCC is crucial. In an in vitro coculture system, differentiated adipocytes were found to enhance cancer stemness and drug resistance in HCC through paracrine signaling.
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