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Guanglin Zhong

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Articles

BCL9 inhibition promotes fibroblast lipogenesis by regulating macrophage–fibroblast interactions to attenuate pulmonary fibrosis

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By Wenjie Wang, YUAN ZHANG, Fenglian He, Li Sun, Huiyu Li, Rongchen Liu, Ling Zhang, Anqi Li, Mei Feng, Yuxuan Dong, Xiaoxuan Lu, Xiaojin Wang, Yuan Si, Yejun Wu, Mengmeng Zhao, Dafu Zhu, Jian Chen, JING CHEN, Ming-Wei Wang, Di Zhu, Likun Gong, Bingshun Wang, Guanglin Zhong, Zhuyi Xi
| Nature Verified
Abstract Idiopathic pulmonary fibrosis (IPF) is a progressive and fatal interstitial lung disease with an urgent need for novel therapeutic strategies. M2 macrophage-derived TGF-β1 promotes fibroblast myogenesis, contributing to IPF pathogenesis. Targeting macrophage polarization and fibroblast function thus represents an effective therapeutic approach for treating IPF. Here, we identify B-cell lymphoma 9 (BCL9) as a key upstream regulator implicated in IPF pathogenesis.
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