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Hyungjin Eoh

As seen in: Nature, Cell Press, Lungcast
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Articles

KSHV vIRF3–PKM2 interaction induces SMAD5 acetylation, promoting Kaposi’s sarcoma progression

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By Jae Lee, Abhishek Kumar, Junjie Zhang, Soochi Kim, Han-Jun Kim, Kyeong Kyu Kim, Zsolt Toth, Hyungjin Eoh, Kyung-Chul Choi, Hye-Ra Lee, Yeong Jun Kim, Juhyeon Lim, Shrute Kannappan
| Nature Verified
Abstract Kaposi’s sarcoma (KS), induced by Kaposi’s sarcoma-associated herpesvirus (KSHV), is a multicentric tumor of lymphatic endothelial origin. However, the fundamental molecular mechanisms underlying the KS progression remain poorly understood.
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NOTCH localizes to mitochondria through the TBC1D15-FIS1 interaction and is stabilized via blockade of E3 ligase and CDK8 recruitment to reprogram tumor-initiating cells | Experimental & Molecular Medicine

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By Yicheng Zhu, Jae-Jin Lee, Hyungjin Eoh, Stanley M. Tahara, Lin Chen
| Nature Verified
Abstract The P53-destabilizing TBC1D15-NOTCH protein interaction promotes self-renewal of tumor-initiating stem-like cells (TICs); however, the mechanisms governing the regulation of this pathway have not been fully elucidated. Here, we show that TBC1D15 stabilizes NOTCH and c-JUN through blockade of E3 ligase and CDK8 recruitment to phosphodegron sequences.
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New Insights into Treating Tuberculosis

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By Hyungjin Eoh
| Lungcast
Dr. Robert Koch, a German physician scientist, discovered Mycobacterium tuberculosis bacilli as a causative agent of TB over 130 years ago. Dr. Koch’s discovery of M. tuberculosis inspired scientists to initiate the antibiotic era to stop the spread of TB. Dr. Schatz and Dr. Waksman discovered an antibiotic called streptomycin in 1944 which showed impressive results when its anti-TB effect using animal models were examined.
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