Irina Petrache on Muck Rack

Irina Petrache

Denver
As seen in: Color Research & Application, Thorax, JAMA (The Journal of the American Medical Association), The Journal of Clinical Investigation, Texas Medical Center, The Journal of Immunology, Respiratory Medicine, touchRESPIRATORY
Chief PCCSM, Wollowick Chair #COPD #research, AVP Faculty Development, Professor of Medicine @njhealth; @CU_PSCCM ; President @ATScommunity ; @the_asci @A_P_S_A
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Articles

"Does Chronic Obstructive Pulmonary Disease Originate from Different Ce" by Yohannes Tesfaigzi, Jeffrey L Curtis et al.

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By Yohannes Tesfaigzi, Jeffrey Curtis, Irina Petrache, Francesca Polverino
| Texas Medical Center
Keywords Humans, Endothelial Cells, Genome-Wide Association Study, Proteomics, Pulmonary Disease, Chronic Obstructive, Lung, lung cell types, single nucleotide polymorphisms, single-cell transcriptomics, lineage tracing, gene-and-environment interaction Abstract The onset of chronic obstructive pulmonary disease (COPD) is heterogeneous, and current approaches to define distinct disease phenotypes are lacking.
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Reduced Bik expression drives low-grade airway inflammation and increased risk for COPD in females

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By Irina Petrache, David Riches, Yohannes A. Mebratu, Scott Randell
| The Journal of Clinical Investigation
Abstract Chronic low-grade inflammation is increasingly recognized as a subtle yet potent risk factor for a multitude of age-related disorders, including respiratory diseases, cardiovascular conditions, metabolic syndromes, autoimmunity, and cancer. In this issue of the JCI, Mebratu, Jones, and colleagues shed new light on the mechanisms that promote low-grade airway inflammation and how this contributes to the development of chronic obstructive pulmonary disease (COPD).
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Marked elevations in lung and plasma ceramide in COVID-19 linked to microvascular injury

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By JCI Insight, Irina Petrache, Elisabet Pujadas, Aditya Ganju
| The Journal of Clinical Investigation
Abstract The pathogenesis of the marked pulmonary microvasculature injury, a distinguishing feature of COVID-19 acute respiratory distress syndrome (COVID-ARDS), remains unclear. Implicated in the pathophysiology of diverse diseases characterized by endothelial damage, including ARDS and ischemic cardiovascular disease, ceramide and in particular palmitoyl ceramide (C16:0-ceramide) may be involved in the microvascular injury in COVID-19.
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