Jeha Jeon on Muck Rack

Jeha Jeon

As seen in: Nature, Cell Reports, Cell Stem Cell
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Articles

Pre-clinical safety and efficacy of human induced pluripotent stem cell-derived products for autologous cell therapy in Parkinson’s disease

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By Jeha Jeon, In-Hee Lee, Heejin Jang, Sanghyeok Ko, Serhiy Naumenko, Minseon Kim, Hannah Ryu, Zenith Shrestha, Nayeon Lee, Tae-Yoon S. Park, HoeWon Park, Seo-Hyun Kim, Ki-Jun Yoon, Jeffrey S. Schweitzer, Todd Herrington, Sek Won Kong, Bob Carter, Pierre Leblanc, Kwang-Soo Kim
| Cell Stem Cell Verified
Keywords pre-clinical study human induced pluripotent stem cells midbrain dopaminergic cells autologous cell therapy Parkinson's disease safety and efficacy genome integrity inter-individual variability dopaminergic fiber density phase 1 clinical trial Get full text access Log in, subscribe or purchase for full access. References 1. Bloem, B.R. ∙ Okun, M.S. ∙ Klein, C. Parkinson's disease Lancet. 2021; 397:2284-2303 2. Kalia, L.V. ∙ Lang, A.E. Parkinson's disease Lancet. 2015; 386:896-912 3.
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Co-transplantation of autologous Treg cells in a cell therapy for Parkinson’s disease - Nature

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By Tae-Yoon S. Park, Jeha Jeon, Nayeon Lee, Dongxin Liu, Minseon Kim, Pierre Leblanc, Jeffrey S. Schweitzer
| Nature Verified
Abstract The specific loss of midbrain dopamine neurons (mDANs) causes major motor dysfunction in Parkinson’s disease, which makes cell replacement a promising therapeutic approach1,2,3,4. However, poor survival of grafted mDANs remains an obstacle to successful clinical outcomes5,6,7,8.
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SIRT2 regulates mitochondrial dynamics and reprogramming via MEK1-ERK-DRP1 and AKT1-DRP1 axes

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By TaeWoo Kim, Jeha Jeon, Yongwoo Jang, Patrick Kim
| Cell Reports Verified
Highlights • SIRT2 regulates DRP1-dependent mitochondrial remodeling in human fibroblasts • SIRT2KD acetylates MEK1 and activates DRP1-dependent mitochondrial remodeling • SIRT2KD acetylates AKT1, leading to DRP1-dependent mitochondrial remodeling • SIRT2 regulates human somatic cell reprogramming via MEK1-ERK and AKT1 axes Summary During somatic reprogramming, cellular energy metabolism fundamentally switches from predominantly mitochondrial oxidative phosphorylation toward glycolysis.
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