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Correction to: Nature https://doi.org/10.1038/s41586-025-09825-y Published online 10 December 2025 It has been drawn to our attention that some statements in the initially published version of this article could be interpreted as supporting a direct mechanistic role for ZNF695 in L1 regulation. As there was no experimental evidence for such a role, the article has been corrected.
Abstract Understanding lung cancer evolution can identify tools for intercepting its growth1,2. Here, in a landscape analysis of 1,024 lung adenocarcinomas (LUADs) with deep whole-genome sequencing integrated with multiomic data, we identified 542 LUADs with a diverse clonal architecture. In this group, we observed divergent evolutionary trajectories based on tobacco smoking exposure, ancestry and sex.
1Laboratory of Translational Genomics, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD, USA 2Laboratory of Genetic Susceptibility, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD, USA 3Integrative Tumor Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Bethesda, MD, USA 4Genome Modification Core, Frederick National Lab for Cancer Research, Frederick, MD, USA 5Institute of...
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