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Juhyun Kim

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Articles

Engineering environmental bacteria for whole-cell PET hydrolysis and assimilation

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By Alice Banks, Umar Abdulmutalib, Christian Sonnendecker, Harry Doherty, Zheren Zhang, Juhyun Kim, Stuart Brown, Ren Wei, Carolina Alvarez-Ortega, Claudio Avignone-Rossa, Gerald Larrouy-Maumus, Wolfgang Zimmermann, Jose Jimenez, José Jiménez Verified, Brooke Wain, Charlotte Bosomworth, Pablo Pomposiello
| Cell Press Verified
Research ArticleOnline nowOpen access Affiliations & Notes 1Department of Life Sciences, Imperial College London, South Kensington, London SW7 2AZ, UK 2Discipline of Microbes, Infection and Immunity, School of Biosciences, Faculty of Health and Medical Sciences, University of Surrey, Guildford GU2 7XH, UK 3Department of Biological Sciences, Faculty of Science, Northwest University, Kano, Muhammad Buhari Way, PM 3220 Kabuga, Kano State, Nigeria 4Imperial Centre for Engineering Biology,...
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TWIK-1 plays distinct roles in spinal and peripheral sensory circuits controlling mechanical sensitivity and neuropathic hypersensitivity

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By Yunjeong Lee, Seojung Kim, Eun Yeong Lim, Junhee Park, Yong Kim, Jonghoon Jung, Yan Zhang, Hyeonwi Son, John Shannonhouse, Juhyun Kim, Eun-Mi Hwang, Byung-Chang Suh, Pojeong Park, Myungin Baek, Jaekwang Lee, Yu Shin Kim, Jae-Yong Park, Hyosang Lee, Tery Yun, Jae Hyuk Jeon, Jin-Nyeong Woo, Eunsil Cho, Mi-Ryoung Song
| Nature Verified
Abstract Ion channels are essential for sensing somatic stimuli, and their dysregulation can cause chronic conditions such as neuropathic pain. Although the potassium channel Twik-1 has been linked to sensory processing, its specific roles in the somatosensory system under normal and disease conditions remain unclear.
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Dysregulated GluA2-Y876 phosphorylation contributes to loss of synaptic upscaling in GRIP1 mutant mice with reduced sociability and increased repetitive behavior | Molecular Psychiatry

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By Rebeca Mejías, Juhyun Kim, Richard Huganir
| Nature Verified
Abstract Loss of synaptic upscaling, a post-synaptic homeostatic plasticity, has been reported in mouse models of autism, but the underlying mechanism remains unknown. Glutamate receptor interacting protein 1 (GRIP1) binds AMPA receptor 2 (GluA2) through its PDZ domains 4-6 where gain-of-function variants were described in autism. We characterized mice carrying one variant, GRIP1-I586L (murine I507L), that shows increased binding with GluA2.
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