Skip To Main Content
Lara Barrio on Muck Rack

Lara Barrio

As seen in: EurekAlert!, Cell Press
Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work. Claim your profile

Get in touch with Lara

Contact Lara, search articles and posts on X, monitor coverage, and track replies from one place.

Learn more about Muck Rack

Actions

Share this page

Is this you?

As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile

Articles

Chromosomal instability-induced cell invasion through caspase-driven DNA damage

|
By Lara Barrio, Baldiri Reixac, Mariana Muzzopappa, Daniela Romão
| Cell Press Verified
These cookies are necessary for the website to function and cannot be switched off in our systems. They are usually only set in response to actions made by you which amount to a request for services, such as setting your privacy preferences, logging in or filling in forms. You can set your browser to block or alert you about these cookies, but some parts of the site will not then work. These cookies do not store any personally identifiable information.
Open in Who Shared View Duplicates (681) Issue with article?

Proteostasis failure and mitochondrial dysfunction leads to aneuploidy-induced senescence

|
By Lara Barrio, Daniela Romão, Jery Joy, Celia Santos-Tapia
| Cell Press Verified
Highlights • Protein quality control mechanisms are near saturation in aneuploid cells • Mitochondrial dysfunction and impaired mitophagy contribute to ROS production • Autophagy or mitophagy activation dampens the deleterious effects of CIN • JNK promotes a senescent state that drives compensatory proliferation and proteostasis Summary Aneuploidy, an unbalanced number of chromosomes, is highly deleterious at the cellular level and leads to senescence, a stress-induced response characterized...
Open in Who Shared Issue with article?

The Upd3 cytokine couples inflammation to maturation defects in Drosophila

|
By Baldiri Reixac, Mariana Muzzopappa, Lara Barrio, Daniela Romão
| Cell Press Verified
Highlights • Highly inflammatory epithelial tumors cause a strong developmental delay • Upd3 cytokine produced by malignant and nearby cells signals to the prothoracic gland • JAK/STAT signal amplifies the systemic levels of Upd3 • JAK/STAT signaling in the ecdysone-producing compartment impacts on the bantam miRNA Summary Developmental transitions, such as puberty or metamorphosis, are tightly controlled by steroid hormones and can be delayed by the appearance of growth abnormalities,...
Open in Who Shared Issue with article?
See all 6 articles