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Mafalda Oliveira

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Articles

Comprehensive clinical cancer analysis of homologous recombination deficiency biomarkers in an academic molecular profiling program

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By Sara Marisa Simonetti, Stephen Pettitt, Paula Proszek, Cristina Viaplana, Lorena Magraner-Pardo, Eduardo García-Galea, Carmen Garcia-Duran, Marcel Romey, Sara Arce-Gallego, Anna Suñol, Flaminia Pedretti, Andrea Herencia-Ropero, Paolo Nuciforo, Mafalda Oliveira, Cristina Saura, Isabel Pimentel, Carsten Denkert, Christopher Lord Verified, Andrew Tutt, Aleix Prat, Rodrigo Dientsmann, Susana Aguilar, Sara Gutiérrez-Enríquez, Joaquin Mateo, Ana Oaknin, Judith Balmaña, Alba Llop-Guevara, Ana Vivancos, Violeta Serra, Heura Domènech, Paula Romero-Lozano, Michaela Smalley, Adela Rodríguez-Hernández, Joanna Domènech-Vivó
| Nature Verified
Abstract PARP inhibitors (PARPi) are effective in tumors with homologous recombination repair (HRR) deficiency (HRD), typically identified by germline/tumor mutations. However, genetic testing may miss intrinsic PARPi sensitivity and resistance. We evaluated strategies to improve detection and longitudinal monitoring of HRD across >500 tumor samples and >20 paired liquid biopsies, integrating genetic, genomic, and functional readouts.
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Clinical audit in improving the quality of care for people with feeding ostomy

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By Bárbara Costa, Mafalda Oliveira, Ana Maria Reis
| Dialnet - Universidad de La Rioja
español Introducción: La provisión de cuidados de enfermería de alta calidad a personas con ostomía alimentaria es un desafío clínico que requiere prácticas sistematizadas, formación continua y seguimiento de los resultados. La auditoría clínica es una estrategia estructurada para la evaluación y mejora continua de la calidad de la atención.
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Exploiting androgen receptor agonism as a treatment strategy in estrogen receptor-positive metastatic breast cancer

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By Laia Monserrat, Cristina E. Molina, Andreu Òdena, Irene Agustí-Barea, Cristina Viaplana, Aerica Nagornyuk, Marta Guzman, Olga Rodriguez, Fara Brasó-Maristany, Eduardo García-Galea, Rodrigo Dienstmann, Motoki Takaku, Aleix Prat, Sarat Chandarlapaty, Julia Ponomarenko, Cristina Saura, Mafalda Oliveira, Lara Nonell, Meritxell Bellet, Violeta Serra, Judith García-García, Taras Yuziv Duda, Gilles Flamen, Ruth Maldonado-Padrol, Claudia Yáñez-Bartolomé, Flaminia Pedretti, Guillermo P. Vicent
| Nature Verified
Abstract The androgen receptor (AR) is expressed in 75% of estrogen receptor-positive (ER+) breast cancers (BC). Selective AR modulators (SARMs), like EP0062, present a promising therapeutic strategy for ER + BC, particularly in patients who cannot tolerate endocrine therapy (ET) or whose tumors have developed resistance. We aimed to study the antitumor activity of EP0062 in ER+ patient-derived xenograft (PDX) models.
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