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Meng-Ting Yin

As seen in: Nature, Cell Reports
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Articles

Exercise-induced anti-obesity effects in male mice generated by a FOXO1-KLF10 reinforcing loop promoting adipose lipolysis - Nature Communications

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By Jie-Ying Zhu, Min Chen, Wang-Jing Mu, Hong-Yang Luo, Shan Li Verified, Lin-Jing Yan, Ruo-Ying Li, Meng-Ting Yin, Xin Li Verified, Hu-Min Chen
| Nature Verified
Abstract Exercise combats obesity and metabolic disorders, but the underlying mechanism is incompletely understood. KLF10, a transcription factor involved in various biological processes, has an undefined role in adipose tissue and obesity. Here, we show that exercise facilitates adipocyte-derived KLF10 expression via SIRT1/FOXO1 pathway.
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Maternal exercise prevents metabolic disorders in offspring mice through SERPINA3C - Nature Metabolism

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By Ruo-Ying Li, Jie-Ying Zhu, Min Chen, Wang-Jing Mu, Hong-Yang Luo, Shan Li Verified, Lin-Jing Yan, Meng-Ting Yin, Xin Li Verified, Hu-Min Chen
| Nature Verified
Abstract Maternal exercise can improve the metabolic health of the offspring. However, the molecular mechanisms underlying the beneficial effects of maternal exercise on the offspring remain unclear.
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Exercise-induced adipokine Nrg4 alleviates MASLD by disrupting hepatic cGAS-STING signaling

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By Min Chen, Jie-Ying Zhu, Wang-Jing Mu, Hong-Yang Luo, Lin-Jing Yan, Shan Li Verified, Ruo-Ying Li, Xin Li, Liang Guo, Meng-Ting Yin, Hu-Min Chen
| Cell Reports Verified
Keywords exercise Nrg4 Erbb4 AKT cGAS STING PPARγ inflammation MASLD hepatic steatosis Research topic(s) CP: Immunology CP: Metabolism Introduction Exercise is an effective way to prevent metabolic disease, and it exerts its protective effects by improving the metabolic phenotypes of multiple tissues.1,2,3,4 These multi-tissue adaptations occur not only through exercise-triggered intrinsic signaling events in each tissue but also through exercise-induced integration of inter-tissue...
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